BRG-1 and brm genes expression during the phenotypic reversion of C6/ST1 glioma cells by glucocorticoid treatment (2000)
- Authors:
- Autor USP: SOGAYAR, MARI CLEIDE - IQ
- Unidade: IQ
- Subjects: BIOQUÍMICA; BIOLOGIA MOLECULAR; EXPRESSÃO GÊNICA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Resumos
- Conference titles: Reunião Anual da Sociedade Brasileira de Bioquímica e Biologia Molecular
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ABNT
ORTIS, Fernanda e VILA, R. L. e SOGAYAR, Mari Cleide. BRG-1 and brm genes expression during the phenotypic reversion of C6/ST1 glioma cells by glucocorticoid treatment. 2000, Anais.. São Paulo: SBBq, 2000. . Acesso em: 23 abr. 2024. -
APA
Ortis, F., Vila, R. L., & Sogayar, M. C. (2000). BRG-1 and brm genes expression during the phenotypic reversion of C6/ST1 glioma cells by glucocorticoid treatment. In Resumos. São Paulo: SBBq. -
NLM
Ortis F, Vila RL, Sogayar MC. BRG-1 and brm genes expression during the phenotypic reversion of C6/ST1 glioma cells by glucocorticoid treatment. Resumos. 2000 ;[citado 2024 abr. 23 ] -
Vancouver
Ortis F, Vila RL, Sogayar MC. BRG-1 and brm genes expression during the phenotypic reversion of C6/ST1 glioma cells by glucocorticoid treatment. Resumos. 2000 ;[citado 2024 abr. 23 ] - Role of polyoma t antigens in balb-c/3t3 cells growth control deregulation
- Further characterization of balb-3t3 transfectants bearing polyoma t antigen genes
- Controle e subversao da proliferacao
- Glucocorticoid-regulated gene in transformed to normal phenotypic reversion
- T antigens role in polyomavirus transformation: c-myc but not c-fos or c-jun expression is a target for middle t
- Cdna cloning and expression of growth control genes
- Fibroblast growth factor and camp induce the expression of fos and jun proto-oncogenes in pc12 cells
- Molecular bases for the transformed to normal phenotypic reversion induced by glucocorticoid hormoners in rat glioma
- Cloning of differentially expressed genes involved glucocorticoid-induced phenotypic reversion of c6 glioma cells
- Hydrazine derivative - induced proliferation superimposed on citotoxity in cultured moused fibroblasts
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