Opening of mitochondrial 'K POT. +' channels increases ischemic ATP levels by preventing hydrolysis (2002)
- Authors:
- Autor USP: KOWALTOWSKI, ALÍCIA JULIANA - IQ
- Unidade: IQ
- Subjects: BIOQUÍMICA; HIDRÓLISE; MITOCÔNDRIAS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Journal of Bioenergetics and Biomembranes
- ISSN: 0145-479X
- Volume/Número/Paginação/Ano: v. 34, n. 4, p. 285-298, 2002
-
ABNT
BELISLE, Eduardo e KOWALTOWSKI, Alicia Juliana. Opening of mitochondrial 'K POT. +' channels increases ischemic ATP levels by preventing hydrolysis. Journal of Bioenergetics and Biomembranes, v. 34, n. 4, p. 285-298, 2002Tradução . . Acesso em: 17 abr. 2024. -
APA
Belisle, E., & Kowaltowski, A. J. (2002). Opening of mitochondrial 'K POT. +' channels increases ischemic ATP levels by preventing hydrolysis. Journal of Bioenergetics and Biomembranes, 34( 4), 285-298. -
NLM
Belisle E, Kowaltowski AJ. Opening of mitochondrial 'K POT. +' channels increases ischemic ATP levels by preventing hydrolysis. Journal of Bioenergetics and Biomembranes. 2002 ; 34( 4): 285-298.[citado 2024 abr. 17 ] -
Vancouver
Belisle E, Kowaltowski AJ. Opening of mitochondrial 'K POT. +' channels increases ischemic ATP levels by preventing hydrolysis. Journal of Bioenergetics and Biomembranes. 2002 ; 34( 4): 285-298.[citado 2024 abr. 17 ] - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
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