Ischemic preconditioning inhibits mitochondrial respiration, increases H2O2 release and enhances 'K+' transport (2003)
- Authors:
- Autor USP: KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- Subjects: BIOQUÍMICA; ADENOSINA TRIFOSFATO; MITOCÔNDRIAS; ISQUEMIA MIOCÁRDICA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Programa e Resumos
- Conference titles: Reunião Anual da Sociedade Brasileira de Bioquímica e Biologia Molecular
-
ABNT
SILVA, Mirian Mateus da et al. Ischemic preconditioning inhibits mitochondrial respiration, increases H2O2 release and enhances 'K+' transport. 2003, Anais.. São Paulo: SBBq, 2003. . Acesso em: 19 abr. 2024. -
APA
Silva, M. M. da, Sartori, A., Belisle, E., & Kowaltowski, A. J. (2003). Ischemic preconditioning inhibits mitochondrial respiration, increases H2O2 release and enhances 'K+' transport. In Programa e Resumos. São Paulo: SBBq. -
NLM
Silva MM da, Sartori A, Belisle E, Kowaltowski AJ. Ischemic preconditioning inhibits mitochondrial respiration, increases H2O2 release and enhances 'K+' transport. Programa e Resumos. 2003 ;[citado 2024 abr. 19 ] -
Vancouver
Silva MM da, Sartori A, Belisle E, Kowaltowski AJ. Ischemic preconditioning inhibits mitochondrial respiration, increases H2O2 release and enhances 'K+' transport. Programa e Resumos. 2003 ;[citado 2024 abr. 19 ] - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
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