Aorta remodeling responses to hemodynamic stimuli induced by coarctation in rats (2005)
- Authors:
- USP affiliated authors: RAMOS, SIMONE GUSMAO - FMRP ; ELIAS JUNIOR, JORGE - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP ; ROSSI, MARCOS ANTONIO - FMRP
- Unidade: FMRP
- Assunto: CARDIOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Hipertensão
- Volume/Número/Paginação/Ano: v. 8, supl., p. 5, res. 017, 2005
- Conference titles: Congresso da Sociedade Brasileira de Hipertensão
-
ABNT
PRADO, Cibele M. et al. Aorta remodeling responses to hemodynamic stimuli induced by coarctation in rats. Hipertensão. São Paulo: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 26 abr. 2024. , 2005 -
APA
Prado, C. M., Ramos, S. G., Alves Filho, J. C. F., Elias Júnior, J., Cunha, F. de Q., & Rossi, M. A. (2005). Aorta remodeling responses to hemodynamic stimuli induced by coarctation in rats. Hipertensão. São Paulo: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Prado CM, Ramos SG, Alves Filho JCF, Elias Júnior J, Cunha F de Q, Rossi MA. Aorta remodeling responses to hemodynamic stimuli induced by coarctation in rats. Hipertensão. 2005 ; 8 5.[citado 2024 abr. 26 ] -
Vancouver
Prado CM, Ramos SG, Alves Filho JCF, Elias Júnior J, Cunha F de Q, Rossi MA. Aorta remodeling responses to hemodynamic stimuli induced by coarctation in rats. Hipertensão. 2005 ; 8 5.[citado 2024 abr. 26 ] - Wall shear stress and stretch differentially affect aorta remodeling in rats
- Hypertension, laminar flow and normal shear stress or normotension, turbulent flow and low wall shear stress cause distinct aorta remodeling
- Hypertension, laminar flow and normal shear stress or normotension, turbulent flow and low shear stress cause distinct aorta remodeling
- Turbulent flow/low wall shear stress and stretch differentially affect aorta remodeling in rats
- Wall shear stress and stretch differentially affect aorta remodeling in rats
- Turbulent blood flow an essential localizing role in the development of atherosclerotic lesions in experimentally induced hypercholesterolaemia in rats
- Circumferential wall tension due to hypertension plays a pivotal role in aorta remodeling
- Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis
- Chronic inhibition of nitric oxide synthase induces hypertension and cardiomyocyte mitochondrial and myocardial collagen remodelling in the absence of hypertrophy
- Turbulent blood flow plays an essential localizing role in the development of atherosclerotic lesions in experimentally-induced hypercholesterolemia in rats
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
