Cellular mechanisms underlying increased phe-induced contraction in contralateral carotid after balloon catheter (2005)
- Authors:
- USP affiliated authors: RAMALHO, LEANDRA NAIRA ZAMBELLI - FMRP ; OLIVEIRA, ANA MARIA DE - FCFRP
- Unidades: FMRP; FCFRP
- Assunto: FARMACOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Revista Brasileira de Ciências Farmacêuticas
- ISSN: 1516-9332
- Volume/Número/Paginação/Ano: v. 41, supl. 1, p. 241, res. FF030, 2005
- Conference titles: Internacional of Congress of Pharmaceutical Science (CIFARP
-
ABNT
OLIVON, Vânia Cláudia et al. Cellular mechanisms underlying increased phe-induced contraction in contralateral carotid after balloon catheter. Revista Brasileira de Ciências Farmacêuticas. São Paulo: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 14 maio 2024. , 2005 -
APA
Olivon, V. C., Andrade, C. de, Ramalho, L. N. Z., & Oliveira, A. M. de. (2005). Cellular mechanisms underlying increased phe-induced contraction in contralateral carotid after balloon catheter. Revista Brasileira de Ciências Farmacêuticas. São Paulo: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Olivon VC, Andrade C de, Ramalho LNZ, Oliveira AM de. Cellular mechanisms underlying increased phe-induced contraction in contralateral carotid after balloon catheter. Revista Brasileira de Ciências Farmacêuticas. 2005 ; 41 241.[citado 2024 maio 14 ] -
Vancouver
Olivon VC, Andrade C de, Ramalho LNZ, Oliveira AM de. Cellular mechanisms underlying increased phe-induced contraction in contralateral carotid after balloon catheter. Revista Brasileira de Ciências Farmacêuticas. 2005 ; 41 241.[citado 2024 maio 14 ] - The balloon catheter induces an increased to angiotensin II in reactivity on distant from the injury site
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- Angiotensin II vascular Hyperreactivity in contralateral artery after ballon catheter injury: ROS and nitric oxide pathway modulation
- Reactive oxygen species participation on angiotensin II-induced vascular hyper-reactivity in contralateral artery after balloon catheter injury
- Consequences of acute or chronic stress on relaxation induced by angiotensin 1-7 in rat carotid
- Vascular hyper-reactivity following arterial balloon injury: ros and nitric oxide participation on distant and dalayed effects
- The role of endothelium in the increased in vascular hyper-reactivity following arterial ballon injury
- Local TAT-'p27 POT.kip1' fusion protein inhibits cell proliferation in rat carotid arteries
- Perivascular injury leads to a reduction in vascular reactivity of the collared and to an enhancement on contralateral carotid artery of rats
- Vascular hyper-reactivity following arterial ballon injury: ROS and prostanoids participation on distant and dalayed effects
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