Prame as a secondary target for Bcr-Abl-positive leukemia (2006)
- Authors:
- USP affiliated authors: CARVALHO, DERMEVAL DE - FCFRP ; ZAGO, MARCO ANTONIO - FMRP ; CASTRO, FABÍOLA ATTIÉ DE - FCFRP ; MENDES, JOAO GUSTAVO PESSINI AMARANTE - ICB
- Unidades: FCFRP; FMRP; ICB
- Subjects: METABOLISMO; BIOENERGÉTICA
- Language: Inglês
- Imprenta:
- Publisher place: Angra dos Reis
- Date published: 2006
- Source:
- Título do periódico: Program and Abstract Book
- Conference titles: International Cell Death Symposium on
-
ABNT
CARVALHO, Dermeval de et al. Prame as a secondary target for Bcr-Abl-positive leukemia. 2006, Anais.. Angra dos Reis: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo, 2006. . Acesso em: 19 abr. 2024. -
APA
Carvalho, D. de, Leroy, J. M. G., Pereira, W. O., Proto-Siqueira, R., Zanichelli, M. A., Camanho, D., et al. (2006). Prame as a secondary target for Bcr-Abl-positive leukemia. In Program and Abstract Book. Angra dos Reis: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. -
NLM
Carvalho D de, Leroy JMG, Pereira WO, Proto-Siqueira R, Zanichelli MA, Camanho D, Collassanti MD, Alves DL, Hamerschlak N, Zago MA, Castro FA de, Amarante-Mendes JGP. Prame as a secondary target for Bcr-Abl-positive leukemia. Program and Abstract Book. 2006 ;[citado 2024 abr. 19 ] -
Vancouver
Carvalho D de, Leroy JMG, Pereira WO, Proto-Siqueira R, Zanichelli MA, Camanho D, Collassanti MD, Alves DL, Hamerschlak N, Zago MA, Castro FA de, Amarante-Mendes JGP. Prame as a secondary target for Bcr-Abl-positive leukemia. Program and Abstract Book. 2006 ;[citado 2024 abr. 19 ] - Prame as a secondary target for BCR-ABL-positive leukemias
- BCR–ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients
- Conversion of CD95 (Fas) type II into type I signaling by sub-lethal doses of cycloheximide
- Development and characterization of murine models of T lymphoma to investigate the impact of oncogene expression
- Cosmomycin D, an anthracycline more potent than Doxorubicin for induction of tumor cell death, synergize with imatinib mesilate to induce apoptosis in Bcr-Abl-positive cells
- T lymphoma murine models to study the impact of ectopic expression of anti-apoptotic oncogenes
- BH3 mimetics and TKI combined therapy for Chronic Myeloid Leukemia
- Study of pro and anti-apoptotic genes to understand the resistance of apoptosis mediated by Bcr-Abl
- PRAME expression is associated with down-regulation of trail in chronic myeloid leukemia
- BCR-ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients
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