Genomic approach to understand the mechanisms invilved in Bcr-Abl-mediated resistance to apoptosis (2006)
- Authors:
- USP affiliated authors: CASTRO, FABÍOLA ATTIÉ DE - FCFRP ; LOPES, LUCIA ROSSETTI - ICB ; MOREIRA FILHO, CARLOS ALBERTO - ICB ; MENDES, JOAO GUSTAVO PESSINI AMARANTE - ICB
- Unidades: FCFRP; ICB
- Subjects: ONCOPROTEÍNAS; EXPRESSÃO GÊNICA; APOPTOSE
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Haematologica
- ISSN: 0390-6078
- Volume/Número/Paginação/Ano: v. 91, suppl. 1, p. 223, res. 0603, 2006
- Conference titles: Congress of The European Hematology Association
-
ABNT
BUENO-DA-SILVA, A. E. B. et al. Genomic approach to understand the mechanisms invilved in Bcr-Abl-mediated resistance to apoptosis. Haematologica. Pavia: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 24 abr. 2024. , 2006 -
APA
Bueno-da-Silva, A. E. B., Brumatti, G., Castro, F. A. de, Jacysyn, J. F., Lopes, L. R., Tobo, P. R., et al. (2006). Genomic approach to understand the mechanisms invilved in Bcr-Abl-mediated resistance to apoptosis. Haematologica. Pavia: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. -
NLM
Bueno-da-Silva AEB, Brumatti G, Castro FA de, Jacysyn JF, Lopes LR, Tobo PR, Vêncio RZ, Moreira-Filho CA, Okamoto OK, Amarante-Mendes JGP. Genomic approach to understand the mechanisms invilved in Bcr-Abl-mediated resistance to apoptosis. Haematologica. 2006 ; 91 223.[citado 2024 abr. 24 ] -
Vancouver
Bueno-da-Silva AEB, Brumatti G, Castro FA de, Jacysyn JF, Lopes LR, Tobo PR, Vêncio RZ, Moreira-Filho CA, Okamoto OK, Amarante-Mendes JGP. Genomic approach to understand the mechanisms invilved in Bcr-Abl-mediated resistance to apoptosis. Haematologica. 2006 ; 91 223.[citado 2024 abr. 24 ] - Survivin and CIAP-1 gene expression is linked to chronic myeloid leukemia progression and poor response to imatinib
- Survivin and CIAP-1 gene expression is linked to chronic myeloid leukemia progression and poor response to imatinib
- Indoleamine 2,3-dioxygenase expression in chronic myelogenous leukemia patients: association with disease progression
- Expression of pro-apoptotic bid and bik genes is decreased in chronic myeloid leukemia and low levels of bik MRNA are associated with lack of response to imatinib treatment
- Genomic approach to understand the mechanisms involved in BCR-ABL mediated resistance to apoptosis
- Down regulation of Hematopoietic SH2 domain containing (HSH2D) in the progression of the chronic myelogenous leukemia
- Correlation between metabolic alterations and apoptosis resistance confered by BCR-ABL expression
- Conversion of CD95 (Fas) type II into type I signaling by sub-lethal doses of cycloheximide
- Development and characterization of murine models of T lymphoma to investigate the impact of oncogene expression
- Cosmomycin D, an anthracycline more potent than Doxorubicin for induction of tumor cell death, synergize with imatinib mesilate to induce apoptosis in Bcr-Abl-positive cells
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