Tissue protection mediated by mitochondrial 'K IND.+' channels (2006)
- Authors:
- Autor USP: KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- Subjects: ISQUEMIA; MITOCÔNDRIAS; CÁLCIO
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Biochimica et Biophysica Acta - Molecular Basis of Disease
- ISSN: 0925-4439
- Volume/Número/Paginação/Ano: v. 1762, n. 2, p. 202-212, 2006
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ABNT
FACUNDO, Hebert Di Tarso Fernandes e FORNAZARI, Maynara e KOWALTOWSKI, Alicia Juliana. Tissue protection mediated by mitochondrial 'K IND.+' channels. Biochimica et Biophysica Acta - Molecular Basis of Disease, v. 1762, n. 2, p. 202-212, 2006Tradução . . Acesso em: 24 abr. 2024. -
APA
Facundo, H. D. T. F., Fornazari, M., & Kowaltowski, A. J. (2006). Tissue protection mediated by mitochondrial 'K IND.+' channels. Biochimica et Biophysica Acta - Molecular Basis of Disease, 1762( 2), 202-212. -
NLM
Facundo HDTF, Fornazari M, Kowaltowski AJ. Tissue protection mediated by mitochondrial 'K IND.+' channels. Biochimica et Biophysica Acta - Molecular Basis of Disease. 2006 ; 1762( 2): 202-212.[citado 2024 abr. 24 ] -
Vancouver
Facundo HDTF, Fornazari M, Kowaltowski AJ. Tissue protection mediated by mitochondrial 'K IND.+' channels. Biochimica et Biophysica Acta - Molecular Basis of Disease. 2006 ; 1762( 2): 202-212.[citado 2024 abr. 24 ] - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
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