Impaired hypertensive rat aorta relaxation to nitric oxide donors (Terpy) can involve superoxide anion production in aorta (2006)
- Authors:
- USP affiliated authors: SILVA, ROBERTO SANTANA DA - FCFRP ; BENDHACK, LUSIANE MARIA - FCFRP
- Unidade: FCFRP
- Subjects: AORTA; ÓXIDO NÍTRICO
- Language: Inglês
- Imprenta:
- Publisher: FCFRP-USP
- Publisher place: Ribeirão Preto
- Date published: 2006
- Source:
- Título do periódico: CD de Resumos
- Conference titles: Simpósio Internacional de Pós-Graduação e Pesquisa da Faculdade de Ciências Farmacêuticas de Ribeirão Preto-USP - SINPOSPq
-
ABNT
RODRIGUES, Gerson Jhonatan et al. Impaired hypertensive rat aorta relaxation to nitric oxide donors (Terpy) can involve superoxide anion production in aorta. 2006, Anais.. Ribeirão Preto: FCFRP-USP, 2006. . Acesso em: 24 abr. 2024. -
APA
Rodrigues, G. J., Lima, R. G. de, Silva, R. S. da, & Bendhack, L. M. (2006). Impaired hypertensive rat aorta relaxation to nitric oxide donors (Terpy) can involve superoxide anion production in aorta. In CD de Resumos. Ribeirão Preto: FCFRP-USP. -
NLM
Rodrigues GJ, Lima RG de, Silva RS da, Bendhack LM. Impaired hypertensive rat aorta relaxation to nitric oxide donors (Terpy) can involve superoxide anion production in aorta. CD de Resumos. 2006 ;[citado 2024 abr. 24 ] -
Vancouver
Rodrigues GJ, Lima RG de, Silva RS da, Bendhack LM. Impaired hypertensive rat aorta relaxation to nitric oxide donors (Terpy) can involve superoxide anion production in aorta. CD de Resumos. 2006 ;[citado 2024 abr. 24 ] - Decreased vasodilation induced by a new nitric oxide donor in two kidney, one clip hypertensive rats is due to impaired 'K POT +' channel activation
- Relaxation induced by nitrosyl compound is decreased in 2 kidney-1 clip rat aorta
- Vasodilatation induced by a new no donor involves guanylyl cyclase, k+ channels, Cl- channels activation and inhibition of "Ca POT. 2"+ channels
- Vasodilatation induced by nitric oxide donors in potentiated in endothelium intact rat aortic rings
- Vitamina C normaliza efeito de novo doador de óxido nítrico e ativador de canais de 'K POT.+' na aorta de ratos hipertensos renais
- Óxido nítrico (NO) liberado de doadores de NO tem efeito hipotensor e induz relaxamento da aorta de rato devido à ativação de canais K+, guanilato ciclase e Ca-ATPASE do retículo sarcoplasmático
- Vasodilatation by donor is due to inhibition of 'Ca POT. +2' influx
- Vasodilatation induced by atrial natriuretic peptide (ANP) and C-type natriuretic peptide (CNP) in isolated aorta from rats
- Ruthenium-nitrite complex as pro-drug releases NO in a tissue and enzyme-dependent way
- Hipotensão e vasodilatação induzidas pelo doador de NO [RU(TERPY)(BDQ)NO+]3+ (TERPY) são modulados pelo endotélio e estresse oxidativo em ratos espontaneamente hipertensos (SHR)
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