Caveolae dysfunction contributes to impaired relaxation induced by nitric oxide donor in aorta from renal hypertensive rats (2007)
- Authors:
- USP affiliated authors: MOREIRA, JORGE EDUARDO - FMRP ; SILVA, ROBERTO SANTANA DA - FCFRP ; BENDHACK, LUSIANE MARIA - FCFRP
- Unidades: FMRP; FCFRP
- Subjects: ÓXIDO NÍTRICO; AORTA; VASODILATAÇÃO
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Journal of Pharmacology and Experimental Therapeutics
- ISSN: 0022-3565
- Volume/Número/Paginação/Ano: v. 323, n. 3, p. 831-837, 2007
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ABNT
RODRIGUES, G. J. et al. Caveolae dysfunction contributes to impaired relaxation induced by nitric oxide donor in aorta from renal hypertensive rats. Journal of Pharmacology and Experimental Therapeutics, v. 323, n. 3, p. 831-837, 2007Tradução . . Acesso em: 25 abr. 2024. -
APA
Rodrigues, G. J., Restini, C. B., Lunardi, C. N., Moreira, J. E., Lima, R. G. de, Silva, R. S. da, & Bendhack, L. M. (2007). Caveolae dysfunction contributes to impaired relaxation induced by nitric oxide donor in aorta from renal hypertensive rats. Journal of Pharmacology and Experimental Therapeutics, 323( 3), 831-837. -
NLM
Rodrigues GJ, Restini CB, Lunardi CN, Moreira JE, Lima RG de, Silva RS da, Bendhack LM. Caveolae dysfunction contributes to impaired relaxation induced by nitric oxide donor in aorta from renal hypertensive rats. Journal of Pharmacology and Experimental Therapeutics. 2007 ; 323( 3): 831-837.[citado 2024 abr. 25 ] -
Vancouver
Rodrigues GJ, Restini CB, Lunardi CN, Moreira JE, Lima RG de, Silva RS da, Bendhack LM. Caveolae dysfunction contributes to impaired relaxation induced by nitric oxide donor in aorta from renal hypertensive rats. Journal of Pharmacology and Experimental Therapeutics. 2007 ; 323( 3): 831-837.[citado 2024 abr. 25 ] - Cross-talk between the sarcoplasmic reticulum and the mitochondrial calcium handling system may play an important role in the regulation of contraction in anococcygeus smooth muscle
- Decreased number of caveolase play critical in the sensitivity to the nitric oxide in renal hypertensive rats aorta
- Cross-talk between the sarcoplasmic reticulum and the mitochondrial calcium handling systems may play an important role in the regulation of contraction in anococcygeus smooth muscle
- Decreased number of caveolae in endothelial cells impairs the relaxation induced by acetylcholine in hipertensive rat aortas
- Decreased vasodilation induced by a new nitric oxide donor in two kidney, one clip hypertensive rats is due to impaired 'K POT +' channel activation
- Relaxation induced by nitrosyl compound is decreased in 2 kidney-1 clip rat aorta
- Vasodilatation induced by a new no donor involves guanylyl cyclase, k+ channels, Cl- channels activation and inhibition of "Ca POT. 2"+ channels
- Vasodilatation induced by nitric oxide donors in potentiated in endothelium intact rat aortic rings
- Vitamina C normaliza efeito de novo doador de óxido nítrico e ativador de canais de 'K POT.+' na aorta de ratos hipertensos renais
- Óxido nítrico (NO) liberado de doadores de NO tem efeito hipotensor e induz relaxamento da aorta de rato devido à ativação de canais K+, guanilato ciclase e Ca-ATPASE do retículo sarcoplasmático
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