Nox 5 expression is regulated by endothelin-1, angiotensin II, and aldosterone in human microvascular endothelial cells and may be upregulated in vascular smooth muscle cells from hypertensive patients (2007)
- Authors:
- Autor USP: PASSAGLIA, RITA DE CASSIA ALEIXO TOSTES - ICB
- Unidade: ICB
- Assunto: FARMACOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Hypertension
- Volume/Número/Paginação/Ano: v. 50, Supplement 4, 2007
- Conference titles: Annual High Blood Pressure Research Conference
-
ABNT
MONTEZANO, Augusto C. et al. Nox 5 expression is regulated by endothelin-1, angiotensin II, and aldosterone in human microvascular endothelial cells and may be upregulated in vascular smooth muscle cells from hypertensive patients. Hypertension. Dallas: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 18 abr. 2024. , 2007 -
APA
Montezano, A. C., Chignalia, A., Callera, G. E., Yogi, A., Passaglia, R. de C. A. T., & Touyz, R. M. (2007). Nox 5 expression is regulated by endothelin-1, angiotensin II, and aldosterone in human microvascular endothelial cells and may be upregulated in vascular smooth muscle cells from hypertensive patients. Hypertension. Dallas: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Montezano AC, Chignalia A, Callera GE, Yogi A, Passaglia R de CAT, Touyz RM. Nox 5 expression is regulated by endothelin-1, angiotensin II, and aldosterone in human microvascular endothelial cells and may be upregulated in vascular smooth muscle cells from hypertensive patients. Hypertension. 2007 ; 50[citado 2024 abr. 18 ] -
Vancouver
Montezano AC, Chignalia A, Callera GE, Yogi A, Passaglia R de CAT, Touyz RM. Nox 5 expression is regulated by endothelin-1, angiotensin II, and aldosterone in human microvascular endothelial cells and may be upregulated in vascular smooth muscle cells from hypertensive patients. Hypertension. 2007 ; 50[citado 2024 abr. 18 ] - ET-1-induced oxidative stress in doca-salt hypertension involves NAD(P)H oxidase-independent mechanisms
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