Redox properties of ATP-sensitive 'K POT. +' channels in brain mitochondria (2007)
- Authors:
- Autor USP: KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- Subjects: MITOCÔNDRIAS; BIOQUÍMICA
- Language: Inglês
- Imprenta:
- Publisher: Facultad de Medicina/Departamento de Bioquímica
- Publisher place: Montevideo
- Date published: 2007
- Source:
- Título do periódico: Abstracts
- Conference titles: Free Radicals in Montevideo
-
ABNT
FORNAZARI, Maynara et al. Redox properties of ATP-sensitive 'K POT. +' channels in brain mitochondria. 2007, Anais.. Montevideo: Facultad de Medicina/Departamento de Bioquímica, 2007. . Acesso em: 26 abr. 2024. -
APA
Fornazari, M., Paula, J. G. de, Castilho, R. F., & Kowaltowski, A. J. (2007). Redox properties of ATP-sensitive 'K POT. +' channels in brain mitochondria. In Abstracts. Montevideo: Facultad de Medicina/Departamento de Bioquímica. -
NLM
Fornazari M, Paula JG de, Castilho RF, Kowaltowski AJ. Redox properties of ATP-sensitive 'K POT. +' channels in brain mitochondria. Abstracts. 2007 ;[citado 2024 abr. 26 ] -
Vancouver
Fornazari M, Paula JG de, Castilho RF, Kowaltowski AJ. Redox properties of ATP-sensitive 'K POT. +' channels in brain mitochondria. Abstracts. 2007 ;[citado 2024 abr. 26 ] - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
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