Acetylcholine induces relaxation dependent on nitric oxide synthase (NOS) and an alternative pathway during sepsis (2008)
- Authors:
- USP affiliated authors: BENDHACK, LUSIANE MARIA - FCFRP ; VERCESI, JULIANA APARECIDA - FCFRP
- Unidade: FCFRP
- Subjects: SEPSE; ÓXIDO NÍTRICO
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Basic and Clinical Pharmacology and Toxicology
- ISSN: 1742-7851
- Volume/Número/Paginação/Ano: v. 102, suppl. 1, p. 39, res. P06-04, 2008
- Conference titles: International EDHF Symposium
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ABNT
ARAÚJO, A. V. et al. Acetylcholine induces relaxation dependent on nitric oxide synthase (NOS) and an alternative pathway during sepsis. Basic and Clinical Pharmacology and Toxicology. Oxford: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 19 abr. 2024. , 2008 -
APA
Araújo, A. V., Ferezin, C. Z., Bonaventura, D., Lunardi, C. N., Rodrigues, G. J., Vercesi, J. A., & Bendhack, L. M. (2008). Acetylcholine induces relaxation dependent on nitric oxide synthase (NOS) and an alternative pathway during sepsis. Basic and Clinical Pharmacology and Toxicology. Oxford: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. -
NLM
Araújo AV, Ferezin CZ, Bonaventura D, Lunardi CN, Rodrigues GJ, Vercesi JA, Bendhack LM. Acetylcholine induces relaxation dependent on nitric oxide synthase (NOS) and an alternative pathway during sepsis. Basic and Clinical Pharmacology and Toxicology. 2008 ; 102 39.[citado 2024 abr. 19 ] -
Vancouver
Araújo AV, Ferezin CZ, Bonaventura D, Lunardi CN, Rodrigues GJ, Vercesi JA, Bendhack LM. Acetylcholine induces relaxation dependent on nitric oxide synthase (NOS) and an alternative pathway during sepsis. Basic and Clinical Pharmacology and Toxicology. 2008 ; 102 39.[citado 2024 abr. 19 ] - Acetylcholine induces relaxation dependent on nitric oxide synthase and cyclooxigenase during sepsis
- Vascular smooth muscle relaxation induced by nitric oxide(NO) donor
- Endothelium negatively modulates the vascular relaxation induced by nitric oxide donor, due to uncoupling NO synthase
- The contribution of guanylyl cyclase and potassium channels to the relaxation induced by the NO donors [Ru(terpy)(bdq)NO+]3+ (Terpy) and sodium nitroprusside (SNP) in rat mesenteric resistance arteries
- Nitric oxide (NO) released form NO donors has hypotensive effect and induces rat aorta relaxation due to the activation of K+ channels, guanylyl-cyclase and SERCA
- The nitric oxide (NO) donors [Ru(terpy)(bdq)NO+]3+(TERPY) and sodium nitroprusside (SNP) have different cellular effects in rat basilar arteries relaxation
- Cellular mechanisms involved in the venodilation induced by nitric oxide donors
- Prostacyclin, not only nitric oxide, is a mediator of the vasorelaxation induced by acetylcholine in aortas from rats submitted to cecal ligation and perforation (CLP)
- Molecular mechanisms involved in the dual blockade of the renin angiotensin system (RAS) on the left ventricular remodeling in renal hypertensive rats (2K-1C)
- Espécies reativas de oxigênio podem modular negativamente o relaxamento induzido pelo nitroprussiato de sódio em aortas de ratos hipertensos
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