Redox properties of the ATP-sensitive 'K POT. +' channel in brain mitochondria (2008)
- Authors:
- Autor USP: KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- Subjects: MITOCÔNDRIAS; BIOQUÍMICA; ISQUEMIA; CÁLCIO
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: FEBS Journal
- ISSN: 1742-464X
- Volume/Número/Paginação/Ano: v. 275, supl. 1, p. 401 res. PP8-58, 2008
- Conference titles: FEBS Congress
-
ABNT
FORNAZARI, Maynara et al. Redox properties of the ATP-sensitive 'K POT. +' channel in brain mitochondria. FEBS Journal. Oxford: Instituto de Química, Universidade de São Paulo. . Acesso em: 23 abr. 2024. , 2008 -
APA
Fornazari, M., Paula, J. G. de, Castilho, R. F., & Kowaltowski, A. J. (2008). Redox properties of the ATP-sensitive 'K POT. +' channel in brain mitochondria. FEBS Journal. Oxford: Instituto de Química, Universidade de São Paulo. -
NLM
Fornazari M, Paula JG de, Castilho RF, Kowaltowski AJ. Redox properties of the ATP-sensitive 'K POT. +' channel in brain mitochondria. FEBS Journal. 2008 ; 275 401 res. PP8-58.[citado 2024 abr. 23 ] -
Vancouver
Fornazari M, Paula JG de, Castilho RF, Kowaltowski AJ. Redox properties of the ATP-sensitive 'K POT. +' channel in brain mitochondria. FEBS Journal. 2008 ; 275 401 res. PP8-58.[citado 2024 abr. 23 ] - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
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- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
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