Human cholesteryl ester transfer expression enhances the mouse survival rate in an experimental systemic inflammation model: a novel role for CETP (2008)
- Authors:
- USP affiliated authors: BARBEIRO, DENISE FREDIANI - FM ; QUINTAO, EDER CARLOS ROCHA - FM ; SORIANO, FRANCISCO GARCIA - FM
- Unidade: FM
- Subjects: CITOCINAS (ANÁLISE); LIPOPROTEÍNAS (ANÁLISE); INFLAMAÇÃO; SEPSE; MACRÓFAGOS
- Language: Inglês
- Imprenta:
- Publisher place: Augusta, GA
- Date published: 2008
- Source:
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ABNT
CAZITA, Patrícia M. et al. Human cholesteryl ester transfer expression enhances the mouse survival rate in an experimental systemic inflammation model: a novel role for CETP. Shock, v. 30, n. 5, p. 590-595, 2008Tradução . . Acesso em: 16 abr. 2024. -
APA
Cazita, P. M., Barbeiro, D. F., Moretti, A. I. S., Quintão, E. C. R., & Soriano, F. G. (2008). Human cholesteryl ester transfer expression enhances the mouse survival rate in an experimental systemic inflammation model: a novel role for CETP. Shock, 30( 5), 590-595. -
NLM
Cazita PM, Barbeiro DF, Moretti AIS, Quintão ECR, Soriano FG. Human cholesteryl ester transfer expression enhances the mouse survival rate in an experimental systemic inflammation model: a novel role for CETP. Shock. 2008 ; 30( 5): 590-595.[citado 2024 abr. 16 ] -
Vancouver
Cazita PM, Barbeiro DF, Moretti AIS, Quintão ECR, Soriano FG. Human cholesteryl ester transfer expression enhances the mouse survival rate in an experimental systemic inflammation model: a novel role for CETP. Shock. 2008 ; 30( 5): 590-595.[citado 2024 abr. 16 ] - Human cetp expression enhances the mouse survival rate by lowering the systemic inflammatory response
- CETP expression enhances the survival rate in a experimental murine systemic inflammation model: a novel role for cholesteryl ester transfer protein
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- Crotoxin modulates inflammation and macrophages?: functions in a murine sepsis model
- Cholesteryl ester transfer protein (CETP) ) transgenic mice present increased survival rate and reduced TLR4 receptor in the liver
- Bone marrow cells transplant in septic mice modulates systemic inflammatory responde via cell-cell contact
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