Treatment of leishmania major lesions with the nitric oxide donor, S-nitrosoglutathione (GSNO) (2008)
- Authors:
- Autor USP: ABRAHAMSOHN, ISES DE ALMEIDA - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher place: Ribeirão Preto
- Date published: 2008
- Source:
- Título do periódico: Resumos
- Conference titles: Congress of the Brazilian Society for Immunology - SBI
-
ABNT
COSTA, Inez Silva Fernandes et al. Treatment of leishmania major lesions with the nitric oxide donor, S-nitrosoglutathione (GSNO). 2008, Anais.. Ribeirão Preto: Instituto de Ciências Biomédicas, Universidade de São Paulo, 2008. . Acesso em: 16 abr. 2024. -
APA
Costa, I. S. F., Souza, G. F. P. de, Oliveira, M. G. de, & Abrahamsohn, I. de A. (2008). Treatment of leishmania major lesions with the nitric oxide donor, S-nitrosoglutathione (GSNO). In Resumos. Ribeirão Preto: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Costa ISF, Souza GFP de, Oliveira MG de, Abrahamsohn I de A. Treatment of leishmania major lesions with the nitric oxide donor, S-nitrosoglutathione (GSNO). Resumos. 2008 ;[citado 2024 abr. 16 ] -
Vancouver
Costa ISF, Souza GFP de, Oliveira MG de, Abrahamsohn I de A. Treatment of leishmania major lesions with the nitric oxide donor, S-nitrosoglutathione (GSNO). Resumos. 2008 ;[citado 2024 abr. 16 ] - Endogenously produced IL-10 and prostaglandins regulate peripheral blood mononuclear cells proliferation to Trypanosoma cruzi antigens
- Nitric oxide produced by macrophages and regulation of resistance to "Trypanosoma cruzi" at the initial phase of infection
- Egulation of Trypanosoma cruzi infection by type interferons
- Il-12 regulation of ifn-gama synthesis in the acute phase of trypanosoma cruzi infection
- IL-12 regulation of IFN-'gama' synthesis in the acute phase of Trypanosoma cruzi infection
- Prostaglandins mediate suppression of lymphoproliferation and cytokine synthesis during the early phase of Trypanosoma cruzi infection
- Il-12 enhances proliferation of peripheral blood mononuclear cells from chagasic patients to trypanosoma cruzi antigen
- Il-10 and ifn-gamma production by spleen cells in trypanosoma cruzi infected mice
- Bordetella pertussis and innate immune response: inhibition of NO production by murine BMDMO through TGF-ß arginase and TLR4
- Immature macrophages derived from mouse bone marrow produce large amounts of IL-12p40 after LPS stimulation
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