Vascular smooth muscle relaxation induced by nitric oxide(NO) donor (2009)
- Authors:
- USP affiliated authors: BENDHACK, LUSIANE MARIA - FCFRP ; VERCESI, JULIANA APARECIDA - FCFRP ; SILVA, ROBERTO SANTANA DA - FCFRP
- Unidade: FCFRP
- Subjects: ÓXIDO NÍTRICO; VASODILATAÇÃO; MÚSCULO LISO VASCULAR
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Abstracts
- Conference titles: International Congress of Physiological Sciences(IUPS)
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ABNT
BENDHACK, Lusiane Maria et al. Vascular smooth muscle relaxation induced by nitric oxide(NO) donor. 2009, Anais.. Nagoya: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo, 2009. . Acesso em: 24 abr. 2024. -
APA
Bendhack, L. M., Rodrigues, G. J., Vercesi, J. A., Lima, R. G. de, Silva, R. S. da, & Bonaventura, D. (2009). Vascular smooth muscle relaxation induced by nitric oxide(NO) donor. In Abstracts. Nagoya: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. -
NLM
Bendhack LM, Rodrigues GJ, Vercesi JA, Lima RG de, Silva RS da, Bonaventura D. Vascular smooth muscle relaxation induced by nitric oxide(NO) donor. Abstracts. 2009 ;[citado 2024 abr. 24 ] -
Vancouver
Bendhack LM, Rodrigues GJ, Vercesi JA, Lima RG de, Silva RS da, Bonaventura D. Vascular smooth muscle relaxation induced by nitric oxide(NO) donor. Abstracts. 2009 ;[citado 2024 abr. 24 ] - Endothelium negatively modulates the vascular relaxation induced by nitric oxide donor, due to uncoupling NO synthase
- The contribution of guanylyl cyclase and potassium channels to the relaxation induced by the NO donors [Ru(terpy)(bdq)NO+]3+ (Terpy) and sodium nitroprusside (SNP) in rat mesenteric resistance arteries
- Nitric oxide (NO) released form NO donors has hypotensive effect and induces rat aorta relaxation due to the activation of K+ channels, guanylyl-cyclase and SERCA
- The nitric oxide (NO) donors [Ru(terpy)(bdq)NO+]3+(TERPY) and sodium nitroprusside (SNP) have different cellular effects in rat basilar arteries relaxation
- Cellular mechanisms involved in the venodilation induced by nitric oxide donors
- Acetylcholine induces relaxation dependent on nitric oxide synthase (NOS) and an alternative pathway during sepsis
- Acetylcholine induces relaxation dependent on nitric oxide synthase and cyclooxigenase during sepsis
- Decreased vasodilation induced by a new nitric oxide donor in two kidney, one clip hypertensive rats is due to impaired 'K POT +' channel activation
- Relaxation induced by nitrosyl compound is decreased in 2 kidney-1 clip rat aorta
- Vasodilatation induced by a new no donor involves guanylyl cyclase, k+ channels, Cl- channels activation and inhibition of "Ca POT. 2"+ channels
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