Essential role of CCR2 in neutrophil tissue infiltration and multiple organ dysfunction in sepsis (2011)
- Authors:
- USP affiliated authors: BASILE FILHO, ANIBAL - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP
- Unidade: FMRP
- Subjects: COAGULAÇÃO; FÍGADO; SEPSE
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: American Journal of Respiratory and Critical Care Medicine
- ISSN: 1073-449X
- Volume/Número/Paginação/Ano: v. 183, n. 2, p. 234-242, 2011
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ABNT
SOUTO, Fabricio O. et al. Essential role of CCR2 in neutrophil tissue infiltration and multiple organ dysfunction in sepsis. American Journal of Respiratory and Critical Care Medicine, v. 183, n. 2, p. 234-242, 2011Tradução . . Acesso em: 19 abr. 2024. -
APA
Souto, F. O., Alves Filho, J. C., Turato, W. M., Martins, M. A., Basile Filho, A., & Cunha, F. de Q. (2011). Essential role of CCR2 in neutrophil tissue infiltration and multiple organ dysfunction in sepsis. American Journal of Respiratory and Critical Care Medicine, 183( 2), 234-242. -
NLM
Souto FO, Alves Filho JC, Turato WM, Martins MA, Basile Filho A, Cunha F de Q. Essential role of CCR2 in neutrophil tissue infiltration and multiple organ dysfunction in sepsis. American Journal of Respiratory and Critical Care Medicine. 2011 ; 183( 2): 234-242.[citado 2024 abr. 19 ] -
Vancouver
Souto FO, Alves Filho JC, Turato WM, Martins MA, Basile Filho A, Cunha F de Q. Essential role of CCR2 in neutrophil tissue infiltration and multiple organ dysfunction in sepsis. American Journal of Respiratory and Critical Care Medicine. 2011 ; 183( 2): 234-242.[citado 2024 abr. 19 ] - "Punctual" determination of cytokines and/or nitric oxide should not be used as a prognostic indicator in severe sepsis
- Failure of neutrophil migration in septic shock
- Falência de migração de neutrófilos e dosagem sérica de citocinas e óxido nítrico da sepse humana
- Failure of neutrophil migration in septic patients
- Choque séptico
- Falência de migração de neutrófilos e dosagem sérica de citocinas e óxido nítrico na sepse humana
- Impaired neutrophil chemotaxis in sepsis associates with GRK expression and inhibition of actin assembly and tyrosine phosphorylation
- Interleukin-33 attenuates sepsis by enhancing neutrophil influx to the site of infection [Carta]
- Neutrophil function in severe sepsis
- Energy expenditure estimated by oxigen consumption determined by thermodilution in mechanically ventilated septic patients
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