Effect of calorie restriction and aging on yeast proteasome activity (2010)
- Authors:
- Autor USP: KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- Assunto: BIOQUÍMICA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Free Radical Biology and Medicine
- ISSN: 0891-5849
- Volume/Número/Paginação/Ano: v. 49, suppl. 1, p. S78 res. 188, 2010
- Conference titles: Annual Meeting of the Society for Free Radical Biology Medicine
-
ABNT
CUNHA, Fernanda Marques da e DEMASI, Marilene e KOWALTOWSKI, Alicia Juliana. Effect of calorie restriction and aging on yeast proteasome activity. Free Radical Biology and Medicine. New York: Instituto de Química, Universidade de São Paulo. . Acesso em: 24 abr. 2024. , 2010 -
APA
Cunha, F. M. da, Demasi, M., & Kowaltowski, A. J. (2010). Effect of calorie restriction and aging on yeast proteasome activity. Free Radical Biology and Medicine. New York: Instituto de Química, Universidade de São Paulo. -
NLM
Cunha FM da, Demasi M, Kowaltowski AJ. Effect of calorie restriction and aging on yeast proteasome activity. Free Radical Biology and Medicine. 2010 ; 49 S78 res. 188.[citado 2024 abr. 24 ] -
Vancouver
Cunha FM da, Demasi M, Kowaltowski AJ. Effect of calorie restriction and aging on yeast proteasome activity. Free Radical Biology and Medicine. 2010 ; 49 S78 res. 188.[citado 2024 abr. 24 ] - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
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