BCR-ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients (2011)
- Authors:
- USP affiliated authors: ZAGO, MARCO ANTONIO - FMRP ; CASTRO, FABÍOLA ATTIÉ DE - FCFRP
- Unidades: FMRP; FCFRP
- DOI: 10.1038/onc.2010.409
- Subjects: CARCINOMA; ADENOCARCINOMA; FARMACOTERAPIA; MELANOMA; NEOPLASIAS MAMÁRIAS; ANTINEOPLÁSICOS
- Language: Inglês
- Imprenta:
- Source:
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
CARVALHO, D. D. de et al. BCR-ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients. Oncogene, v. 30, n. 2, p. 223-233, 2011Tradução . . Disponível em: https://doi.org/10.1038/onc.2010.409. Acesso em: 23 abr. 2024. -
APA
Carvalho, D. D. de, Binato, R., Pereira, W. O., Leroy, J. M. G., Colassanti, M. D., Proto-Siqueira, R., et al. (2011). BCR-ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients. Oncogene, 30( 2), 223-233. doi:10.1038/onc.2010.409 -
NLM
Carvalho DD de, Binato R, Pereira WO, Leroy JMG, Colassanti MD, Proto-Siqueira R, Bueno da Silva AEB, Zago MA, Zanichelli MA, Abdelhay E, Castro FA de, Jacysyn JF, Amarante-Mendes JGP. BCR-ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients [Internet]. Oncogene. 2011 ; 30( 2): 223-233.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1038/onc.2010.409 -
Vancouver
Carvalho DD de, Binato R, Pereira WO, Leroy JMG, Colassanti MD, Proto-Siqueira R, Bueno da Silva AEB, Zago MA, Zanichelli MA, Abdelhay E, Castro FA de, Jacysyn JF, Amarante-Mendes JGP. BCR-ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients [Internet]. Oncogene. 2011 ; 30( 2): 223-233.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1038/onc.2010.409 - PRAME expression is associated with down-regulation of trail in chronic myeloid leukemia
- BCR–ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients
- Increased expression of protease-activated receptor 1 (PAR-1) in human leukemias
- Alterações imunológicas associadas ao uso de interferon-'alfa' na leucemia mielóide
- Overexpression of the anti-apoptotic genes mcl-1, bcl-W, bcl-XL and a1 correlates with CML progression and resistance to Gleevec
- Epigenetic therapy induces transcription of inverted SINEs and ADAR1 dependency
- Pharmacological interference with the apoptosis machinery
- A1, survivin and c-FLIP anti-apoptotic genes overexpression in head and neck squamous cell carcinoma (HNSCC) patients
- Genomic approach to undertand the mechenisms involved in BCR-ABL mediated resistance to apoptosis
- Estímulo da expressão dos antígenos HLA-DR em leucemias
Informações sobre o DOI: 10.1038/onc.2010.409 (Fonte: oaDOI API)
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