Aurora kinases: potential therapeutic targets in K-Ras-induced lung cancer (2012)
- Authors:
- Autor USP: BASSÈRES, DANIELA SANCHEZ - IQ
- Unidade: IQ
- Subjects: BIOQUÍMICA; ONCOGENES
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq)
- Publisher place: São Paulo
- Date published: 2012
- Source:
- Título do periódico: Program and Index
- Conference titles: Annual Meeting of the Brazilian Biochemistry and Molecular Biology Society (SBBq)
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ABNT
OZORIO DOS SANTOS, E e AOKI, M. N e BASSÈRES, Daniela Sanchez. Aurora kinases: potential therapeutic targets in K-Ras-induced lung cancer. 2012, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq), 2012. . Acesso em: 20 abr. 2024. -
APA
Ozorio dos Santos, E., Aoki, M. N., & Bassères, D. S. (2012). Aurora kinases: potential therapeutic targets in K-Ras-induced lung cancer. In Program and Index. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq). -
NLM
Ozorio dos Santos E, Aoki MN, Bassères DS. Aurora kinases: potential therapeutic targets in K-Ras-induced lung cancer. Program and Index. 2012 ;[citado 2024 abr. 20 ] -
Vancouver
Ozorio dos Santos E, Aoki MN, Bassères DS. Aurora kinases: potential therapeutic targets in K-Ras-induced lung cancer. Program and Index. 2012 ;[citado 2024 abr. 20 ] - Targeted BMI1 inhibition impairs tumor growth in lung adenocarcinomas with low CEBP alpha expression
- Exploring IKKβ kinase as a therapeutic target for KRAS-driven lung tumor initiating cells
- Exploring IKKβ as an anti-metastatic therapeutic target in KRAS-induced lung cancer
- Targeting Aurora A kinase activity in lung cancer initiating cells driven by oncogenic KRAS
- IKKβ inhibition suppresses sphere formation and self renewal of lung cancer initiating cells
- Aurora kinase targeting in lung cancer reduces KRAS-induced transformation
- IKKβ kinase promotes stemness, migration, and invasion in KRAS-driven lung adenocarcinoma cells
- Identification of a targetable KRAS-mutant epithelial population in non-small cell lung cancer
- FOXO1 inhibition by oncogenic KRAS as an important mechanism promoting lung oncogenesis
- Requirement of the NF-'capa' B subunit p65/RelA for 'capa'-Ras-induced lung tumorigenesis
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