Different responses of two HNSCC cell lineages to oxidative stress (2012)
- Authors:
- USP affiliated authors: CURTI, CARLOS - FCFRP ; LEOPOLDINO, ANDRÉIA MACHADO - FCFRP
- Unidade: FCFRP
- Subjects: BIOQUÍMICA CELULAR; PROTEÍNAS; ESTRESSE OXIDATIVO; NEOPLASIAS DE CABEÇA E PESCOÇO
- Language: Inglês
- Imprenta:
- Publisher place: Foz do Iguaçu
- Date published: 2012
- Conference titles: Reunião Anual da Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq)
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ABNT
GRASSI, M. L. e LEOPOLDINO, Andréia Machado e CURTI, Carlos. Different responses of two HNSCC cell lineages to oxidative stress. 2012, Anais.. Foz do Iguaçu: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo, 2012. . Acesso em: 19 abr. 2024. -
APA
Grassi, M. L., Leopoldino, A. M., & Curti, C. (2012). Different responses of two HNSCC cell lineages to oxidative stress. In . Foz do Iguaçu: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. -
NLM
Grassi ML, Leopoldino AM, Curti C. Different responses of two HNSCC cell lineages to oxidative stress. 2012 ;[citado 2024 abr. 19 ] -
Vancouver
Grassi ML, Leopoldino AM, Curti C. Different responses of two HNSCC cell lineages to oxidative stress. 2012 ;[citado 2024 abr. 19 ] - SET/I2PP2A is an essential protein for cell migration in head and neck squamous cell carcinoma
- SET protects mitochondria from apoptosis signaling and favors autophagy in HNSCC cells exposed to a pro-oxidant
- Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation
- SET protects mitochondrial function and induces autophagy in response to redox unbalance
- SET protein regulates expression of glutathione S-transferase pi 1 (GSTP-1) in oral carcinoma
- Knockdown of SET protein sensitizes HNSCC-HN12 cells to UCN-01
- ATP production in HnRNP K-overexpressing HEK293 cells
- Maternal protein restriction increases respiratory and sympathetic activities and sensitizes peripheral chemoreflex in male rat offspring
- SET protein accumulation prevents oxidative stress-induced apoptosis/mitochondrial dysfunction with potential implication in tumorigenesis
- Neuroprotective effects of C-Phycocyanin in neuronal cell lines and transient ischemic rat retina
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