Protein quality control disruption by PKCβII in heart failure; rescue by the selective PKCβII inhibitor, βIIV5-3 (2012)
- Authors:
- USP affiliated authors: FERREIRA, JULIO CESAR BATISTA - ICB ; BRUM, PATRICIA CHAKUR - EEFE
- Unidades: ICB; EEFE
- DOI: 10.1371/journal.pone.0033175
- Subjects: INSUFICIÊNCIA CARDÍACA; CARDIOLOGIA
- Language: Inglês
- Imprenta:
- Publisher place: San Francisco
- Date published: 2012
- Source:
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
FERREIRA, Julio Cesar Batista et al. Protein quality control disruption by PKCβII in heart failure; rescue by the selective PKCβII inhibitor, βIIV5-3. PLOS ONE, v. 7, n. 3, p. 1-9, 2012Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0033175. Acesso em: 29 mar. 2024. -
APA
Ferreira, J. C. B., Boer, B. N., Grinberg, M., Brum, P. C., & Mochly-Rosen, D. (2012). Protein quality control disruption by PKCβII in heart failure; rescue by the selective PKCβII inhibitor, βIIV5-3. PLOS ONE, 7( 3), 1-9. doi:10.1371/journal.pone.0033175 -
NLM
Ferreira JCB, Boer BN, Grinberg M, Brum PC, Mochly-Rosen D. Protein quality control disruption by PKCβII in heart failure; rescue by the selective PKCβII inhibitor, βIIV5-3 [Internet]. PLOS ONE. 2012 ; 7( 3): 1-9.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1371/journal.pone.0033175 -
Vancouver
Ferreira JCB, Boer BN, Grinberg M, Brum PC, Mochly-Rosen D. Protein quality control disruption by PKCβII in heart failure; rescue by the selective PKCβII inhibitor, βIIV5-3 [Internet]. PLOS ONE. 2012 ; 7( 3): 1-9.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1371/journal.pone.0033175 - Exercise training prevents myocardial infarction induced-skeletal muscle atrophy by reducing oxidative stress in rats
- Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy
- Protein quality control disruption by PKCbetaII in heart failure
- Exercise training decreases NADPH oxidase activity and restores skeletal muscle mass in heart failure rats
- Exercise training restores cardiac protein quality control in heart failure
- Aerobic exercise training upregulates skeletal muscle calpain and ubiquitin-proteasome systems in healthy mice
- Exercise reestablishes autophagic flux and mitochondrial quality control in heart failure
- Treinamento físico aeróbico reduz estresse oxidativo e hiperativação do sistema ubiquitina proteassoma prevenindo atrofia do músculo esquelético na insuficiência cardíaca
- Increased clearance of reactive aldehydes and damaged proteins in hypertension-induced compensated cardiac hypertrophy: impact of exercise training
- β2-Adrenergic signaling modulates mitochondrial function and morphology in skeletal muscle in response to aerobic exercise
Informações sobre o DOI: 10.1371/journal.pone.0033175 (Fonte: oaDOI API)
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