SET protects mitochondria from apoptosis signaling and favors autophagy in HNSCC cells exposed to a pro-oxidant (2012)
- Authors:
- USP affiliated authors: CURTI, CARLOS - FCFRP ; LEOPOLDINO, ANDRÉIA MACHADO - FCFRP
- Unidade: FCFRP
- Subjects: CARCINOMA DE CÉLULAS ESCAMOSAS; PROTEÍNAS; APOPTOSE
- Language: Inglês
- Imprenta:
- Publisher place: Kidlington
- Date published: 2012
- Source:
- Título do periódico: European Journal of Cancer
- ISSN: 0959-8049
- Volume/Número/Paginação/Ano: v. 48, suppl. 5, p. S148, res. 621, 2012
- Conference titles: Biennial Congress of the European Association for Cancer Research
-
ABNT
PESTANA, C. R. e LEOPOLDINO, Andréia Machado e CURTI, Carlos. SET protects mitochondria from apoptosis signaling and favors autophagy in HNSCC cells exposed to a pro-oxidant. European Journal of Cancer. Kidlington: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 19 abr. 2024. , 2012 -
APA
Pestana, C. R., Leopoldino, A. M., & Curti, C. (2012). SET protects mitochondria from apoptosis signaling and favors autophagy in HNSCC cells exposed to a pro-oxidant. European Journal of Cancer. Kidlington: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. -
NLM
Pestana CR, Leopoldino AM, Curti C. SET protects mitochondria from apoptosis signaling and favors autophagy in HNSCC cells exposed to a pro-oxidant. European Journal of Cancer. 2012 ; 48 S148.[citado 2024 abr. 19 ] -
Vancouver
Pestana CR, Leopoldino AM, Curti C. SET protects mitochondria from apoptosis signaling and favors autophagy in HNSCC cells exposed to a pro-oxidant. European Journal of Cancer. 2012 ; 48 S148.[citado 2024 abr. 19 ] - SET/I2PP2A is an essential protein for cell migration in head and neck squamous cell carcinoma
- Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation
- SET protects mitochondrial function and induces autophagy in response to redox unbalance
- SET protein regulates expression of glutathione S-transferase pi 1 (GSTP-1) in oral carcinoma
- Different responses of two HNSCC cell lineages to oxidative stress
- Knockdown of SET protein sensitizes HNSCC-HN12 cells to UCN-01
- ATP production in HnRNP K-overexpressing HEK293 cells
- Maternal protein restriction increases respiratory and sympathetic activities and sensitizes peripheral chemoreflex in male rat offspring
- SET protein accumulation prevents oxidative stress-induced apoptosis/mitochondrial dysfunction with potential implication in tumorigenesis
- Neuroprotective effects of C-Phycocyanin in neuronal cell lines and transient ischemic rat retina
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