NMDA and AMPA/KAINATE receptors of the prelimbic medial prefrontal cortex modulate the elaborated defensive-behaviour and innate fear-induced antinociception elicited by GABAA receptor blockade in the medial hypothalamus (2012)
- Authors:
- USP affiliated authors: HALLAK, JAIME EDUARDO CECILIO - FMRP ; CRIPPA, JOSÉ ALEXANDRE DE SOUZA - FMRP ; COIMBRA, NORBERTO CYSNE - FMRP
- Unidade: FMRP
- Subjects: COMPORTAMENTO; MEDO; HIPOTÁLAMO
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Abstracts
- Conference titles: World Symposium on Translational Models of Panic Disorder
-
ABNT
FREITAS, R. L. et al. NMDA and AMPA/KAINATE receptors of the prelimbic medial prefrontal cortex modulate the elaborated defensive-behaviour and innate fear-induced antinociception elicited by GABAA receptor blockade in the medial hypothalamus. 2012, Anais.. Vitória: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2012. . Acesso em: 19 abr. 2024. -
APA
Freitas, R. L., Salgado-Rohner, C. J., Biagione, A. F., Medeiros, P., Hallak, J. E. C., Crippa, J. A. de S., & Coimbra, N. C. (2012). NMDA and AMPA/KAINATE receptors of the prelimbic medial prefrontal cortex modulate the elaborated defensive-behaviour and innate fear-induced antinociception elicited by GABAA receptor blockade in the medial hypothalamus. In Abstracts. Vitória: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Freitas RL, Salgado-Rohner CJ, Biagione AF, Medeiros P, Hallak JEC, Crippa JA de S, Coimbra NC. NMDA and AMPA/KAINATE receptors of the prelimbic medial prefrontal cortex modulate the elaborated defensive-behaviour and innate fear-induced antinociception elicited by GABAA receptor blockade in the medial hypothalamus. Abstracts. 2012 ;[citado 2024 abr. 19 ] -
Vancouver
Freitas RL, Salgado-Rohner CJ, Biagione AF, Medeiros P, Hallak JEC, Crippa JA de S, Coimbra NC. NMDA and AMPA/KAINATE receptors of the prelimbic medial prefrontal cortex modulate the elaborated defensive-behaviour and innate fear-induced antinociception elicited by GABAA receptor blockade in the medial hypothalamus. Abstracts. 2012 ;[citado 2024 abr. 19 ] - Cannabidiol-mediated modulation of the defensive behaviour evoked by GABA(A) receptor blockade in the inferior colliculus
- Role of 5HT1A receptor on innate fear behavioural attentuation by cannabidiol in a prey versus predator paradigm
- Involvement of prelimbic medial prefrontal cortex in panic-like elaborated defensive behaviour and innate fear-induced antinociception elicited by 'GABA IND.A' receptor blockade in the dorsomedial and ventromedial hypothalamic nuclei: role of the endocannabinoid C'B IND.1' receptor
- NMDA and AMPA/Kainate glutamatergic receptors in the prelimbic medial prefrontal cortex modulate the elaborated defensive behavior and innate fear-induced antinociception elicited by 'GABA IND.A' receptor blockade in the medial hypothalamus
- Role of medial prefrontal cortex in the GABAergic modulation of elaborated defensive behaviour organised by medial hypothalamus
- Medial prefrontal cortex and CB1 endocannabinoid receptors in the antinociception induced by elaborated escape behaviour caused by disinhibition of hypothalamus
- Anti-aversive effects cannabidiol on innate fear-induced behaviors evoked by an ethological model of panic attacks based on a prey vs the wild snake epicrates cenchria crassus confrontation paradigm
- Dissociation between the panicolytic effect of cannabidiol microinjected into the substantia nigra, pars reticulata, and fear-induced antinociception elicited by bicuculline administration in deep layers of the superior colliculus: the role of CB1-cannabinoid receptor in the ventral mesencephalon
- The role of 5-H'T IND.1A' receptors in the anti-aversive effects of cannabidiol on panic attack-like behaviors evoked in the presence of the wild snake Epicrates cenchria crassus (Reptilia, Boidae)
- Cannabidiol and endogenous opioid peptide-mediated mechanisms modulate antinociception induced by transcutaneous electrostimulation of the peripheral nervous system
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
