Chronic intermittent hypoxia depresses afferent neurotransmission in NTS neurons by a reduction in the number of active synapses (2012)
- Authors:
- USP affiliated authors: MACHADO, BENEDITO HONÓRIO - FMRP ; LEÃO, RICARDO MAURICIO XAVIER - FMRP
- Unidade: FMRP
- DOI: 10.1523/JNEUROSCI.2654-12.2012
- Subjects: NEUROTRANSMISSORES; NEURÔNIOS; SINAPSE
- Language: Inglês
- Imprenta:
- Publisher place: Washington
- Date published: 2012
- Source:
- Título do periódico: The Journal of Neuroscience
- ISSN: 0270-6474
- Volume/Número/Paginação/Ano: v. 32, n. 47, p. 16736-16746, 2012
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: hybrid
- Licença: cc-by-nc-sa
-
ABNT
ALMADO, Carlos Eduardo L. e MACHADO, Benedito Honório e LEÃO, Ricardo Maurício Xavier. Chronic intermittent hypoxia depresses afferent neurotransmission in NTS neurons by a reduction in the number of active synapses. The Journal of Neuroscience, v. 32, n. 47, p. 16736-16746, 2012Tradução . . Disponível em: https://doi.org/10.1523/JNEUROSCI.2654-12.2012. Acesso em: 19 abr. 2024. -
APA
Almado, C. E. L., Machado, B. H., & Leão, R. M. X. (2012). Chronic intermittent hypoxia depresses afferent neurotransmission in NTS neurons by a reduction in the number of active synapses. The Journal of Neuroscience, 32( 47), 16736-16746. doi:10.1523/JNEUROSCI.2654-12.2012 -
NLM
Almado CEL, Machado BH, Leão RMX. Chronic intermittent hypoxia depresses afferent neurotransmission in NTS neurons by a reduction in the number of active synapses [Internet]. The Journal of Neuroscience. 2012 ; 32( 47): 16736-16746.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1523/JNEUROSCI.2654-12.2012 -
Vancouver
Almado CEL, Machado BH, Leão RMX. Chronic intermittent hypoxia depresses afferent neurotransmission in NTS neurons by a reduction in the number of active synapses [Internet]. The Journal of Neuroscience. 2012 ; 32( 47): 16736-16746.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1523/JNEUROSCI.2654-12.2012 - Chronic intermittent hypoxia (CIH) enhances spontaneous synaptic transmission in the nucleus tractus solitarii (NTS) neurons of juvenile rats
- Intrinsic pacemaker activity of pre-sympathetic neurons of adult rats is not altered by chronic intermittent hypoxia (CIH)
- Chronic intermittent hypoxia in rats depressess solitary tract evoked neurotransmision in second-but not in higher-order NTS neurons
- Depressed TS-evoked glutamatergic transmission on NTS neurons of rats submitted to intermittent hypoxia is due to reduced number of functional synapses
- Distinct involvement of adenosine receptors in hypoxia-induced neuronal responses in the Nucleus Tractus Solitarii (NTS)
- Depressed TS-evoked glutamatergic transmission on NTS neurons of CIH rats is due to reduced number of functional synapses
- Depressed TS-evoked glutamatergic transmission on NTS neurons of CIH rats is due to reduced number of functional synapses
- Inhibition of spontaneous neurotransmission in the nucleus of solitary tract of the rat by the cannabinoid agonist WIN 55212-2 is not via CB1 or CB2 receptors
- The cannabinoid agonist win 55,212-2 inhibits spontaneous neurotransmission in neurons from rat nucleus tractus solitarius(nts)
- Depressed TS-evoked glutamatergic transmission on NTS neurons of rats submitted to intermittent hypoxia is due to reduced number of functional synapses
Informações sobre o DOI: 10.1523/JNEUROSCI.2654-12.2012 (Fonte: oaDOI API)
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