In pulmonary Paracoccidioidomycosis IL-10 deficiency leads to increased immunity and regressive infection without enhancing tissue pathology (2013)
- Authors:
- USP affiliated authors: COSTA, TÂNIA ALVES DA - ICB ; CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- DOI: 10.1371/journal.pntd.0002512
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher place: San Francisco
- Date published: 2013
- Source:
- Título do periódico: Plos Neglected Tropical Diseases
- ISSN: 1935-2727
- Volume/Número/Paginação/Ano: v. 7, n. 10, e2512, p. 01-13, 2013
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
COSTA, Tânia Alves et al. In pulmonary Paracoccidioidomycosis IL-10 deficiency leads to increased immunity and regressive infection without enhancing tissue pathology. Plos Neglected Tropical Diseases, v. 7, n. 10, p. 01-13, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pntd.0002512. Acesso em: 13 maio 2024. -
APA
Costa, T. A., Bazan, S. B., Feriotti, C., Araújo, E. F., Bassi, Ê. J., Loures, F. V., & Calich, V. L. G. (2013). In pulmonary Paracoccidioidomycosis IL-10 deficiency leads to increased immunity and regressive infection without enhancing tissue pathology. Plos Neglected Tropical Diseases, 7( 10), 01-13. doi:10.1371/journal.pntd.0002512 -
NLM
Costa TA, Bazan SB, Feriotti C, Araújo EF, Bassi ÊJ, Loures FV, Calich VLG. In pulmonary Paracoccidioidomycosis IL-10 deficiency leads to increased immunity and regressive infection without enhancing tissue pathology [Internet]. Plos Neglected Tropical Diseases. 2013 ; 7( 10): 01-13.[citado 2024 maio 13 ] Available from: https://doi.org/10.1371/journal.pntd.0002512 -
Vancouver
Costa TA, Bazan SB, Feriotti C, Araújo EF, Bassi ÊJ, Loures FV, Calich VLG. In pulmonary Paracoccidioidomycosis IL-10 deficiency leads to increased immunity and regressive infection without enhancing tissue pathology [Internet]. Plos Neglected Tropical Diseases. 2013 ; 7( 10): 01-13.[citado 2024 maio 13 ] Available from: https://doi.org/10.1371/journal.pntd.0002512 - Paracoccidoides brasiliensis 30 kDa adhesin: identification as a 14-3-3 protein, cloning and subcellular localization in infection models
- Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis
- Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis
- Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection
- A função da IL-10 na paracoccidioidomicose pulmonar murina
- Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis
- Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis
- AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection
- In vivo treatment with ril-12 protects susceptible mice from Paracoccidioides brasiliensis infection whereas in vivo neutralization of il-4 does not alter disease outcome
- Invariant natural killer T cells as key players in host resistance against Paracoccidioides brasiliensis
Informações sobre o DOI: 10.1371/journal.pntd.0002512 (Fonte: oaDOI API)
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