Apoptosis-associated speck–like protein containing a caspase recruitment domain inflammasomes mediate IL-1β response and host resistance to Trypanosoma cruzi infection (2013)
- Authors:
- Silva, Grace Kelly da
- Costa, Renata Sesti
- Silveira, Tatiana Nunes
- Caetano, Braulia C. - University of Massachusetts (UMass)
- Horta, Catarina Veltrini
- Gutierrez, Fredy Roberto Salazar
- Guedes, Paulo Marcos da Matta - Universidade Federal do Rio Grande do Norte (UFRN)
- Andrade, Warrison A. - Universidade Federal de Minas Gerais (UFMG)
- De Niz, Mariana
- Gazzinelli, Ricardo Tostes - University of Massachusetts (UMass)
- Zamboni, Dario Simões
- Silva, João Santana da
- USP affiliated authors: ZAMBONI, DARIO SIMÕES - FMRP ; SILVA, JOÃO SANTANA DA - FMRP
- Unidade: FMRP
- DOI: 10.4049/jimmunol.1203293
- Subjects: TRYPANOSOMA CRUZI; IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Journal of Immunology
- ISSN: 0022-1767
- Volume/Número/Paginação/Ano: v. 191, n. 6, p. 3373-3383, 2013
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: bronze
-
ABNT
SILVA, Grace Kelly da et al. Apoptosis-associated speck–like protein containing a caspase recruitment domain inflammasomes mediate IL-1β response and host resistance to Trypanosoma cruzi infection. Journal of Immunology, v. 191, n. 6, p. 3373-3383, 2013Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1203293. Acesso em: 26 abr. 2024. -
APA
Silva, G. K. da, Costa, R. S., Silveira, T. N., Caetano, B. C., Horta, C. V., Gutierrez, F. R. S., et al. (2013). Apoptosis-associated speck–like protein containing a caspase recruitment domain inflammasomes mediate IL-1β response and host resistance to Trypanosoma cruzi infection. Journal of Immunology, 191( 6), 3373-3383. doi:10.4049/jimmunol.1203293 -
NLM
Silva GK da, Costa RS, Silveira TN, Caetano BC, Horta CV, Gutierrez FRS, Guedes PM da M, Andrade WA, De Niz M, Gazzinelli RT, Zamboni DS, Silva JS da. Apoptosis-associated speck–like protein containing a caspase recruitment domain inflammasomes mediate IL-1β response and host resistance to Trypanosoma cruzi infection [Internet]. Journal of Immunology. 2013 ; 191( 6): 3373-3383.[citado 2024 abr. 26 ] Available from: https://doi.org/10.4049/jimmunol.1203293 -
Vancouver
Silva GK da, Costa RS, Silveira TN, Caetano BC, Horta CV, Gutierrez FRS, Guedes PM da M, Andrade WA, De Niz M, Gazzinelli RT, Zamboni DS, Silva JS da. Apoptosis-associated speck–like protein containing a caspase recruitment domain inflammasomes mediate IL-1β response and host resistance to Trypanosoma cruzi infection [Internet]. Journal of Immunology. 2013 ; 191( 6): 3373-3383.[citado 2024 abr. 26 ] Available from: https://doi.org/10.4049/jimmunol.1203293 - Ros-dependent NLRP3-inflammasome activation is critical for restriction of Leishmania amazonensis infection
- Critical role of ASC inflammasomes in host innate resistance to Trypanosoma cruzi infection
- NOD/RIP2 signaling contribute to development of a TH1 response and resistance against L. major infection
- A parent-of-origin effect determines the susceptibility of a non-informative F1 population to Trypanosoma cruzi infection in vivo
- IFN-γ plays a unique role in protection against low virulent Trypanosoma cruzi strain
- Restriction of Leishmania amazonensis infection by the inflammasomes
- Intrinsic expression of Nod2 in CD4+ T lymphocytes is not necessary for the development of cell-mediated immunity and host resistance to Toxoplasma gondii
- Role of NLRC4 inflammasome in Paracoccidioides brasiliensis pulmonary inflammation
- Relevance of the myeloid differentiation factor 88 (MyD88) on RANKL, OPG, and nod expressions induced by TLR and IL-1R signaling in bone marrow stromal cells
- NOD1 and NOD2 receptors account to innate and adaptive immune response against Leishmania infection
Informações sobre o DOI: 10.4049/jimmunol.1203293 (Fonte: oaDOI API)
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