Decreased endothelial nitric oxide, systemic oxidative stress, and increased sympathetic modulation contribute to hypertension in obese rats (2014)
- Authors:
- USP affiliated authors: GRANDO, MARCELLA DARUGE - FCFRP ; BENDHACK, LUSIANE MARIA - FCFRP
- Unidade: FCFRP
- DOI: 10.1152/ajpheart.00520.2013
- Subjects: ÓXIDO NÍTRICO; ENDOTÉLIO; GLUTAMATOS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: American Journal of Physiology Heart and Circulatory Physiology
- ISSN: 0363-6135
- Volume/Número/Paginação/Ano: v. 306, n. 10, p. H1472-H1480, 2014
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: green
- Licença: other-oa
-
ABNT
CUNHA, Natalia Veronez da et al. Decreased endothelial nitric oxide, systemic oxidative stress, and increased sympathetic modulation contribute to hypertension in obese rats. American Journal of Physiology Heart and Circulatory Physiology, v. 306, n. 10, p. H1472-H1480, 2014Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00520.2013. Acesso em: 20 abr. 2024. -
APA
Cunha, N. V. da, Pinge-Filho, P., Panis, C., Silva, B. R., Pernomian, L., Grando, M. D., et al. (2014). Decreased endothelial nitric oxide, systemic oxidative stress, and increased sympathetic modulation contribute to hypertension in obese rats. American Journal of Physiology Heart and Circulatory Physiology, 306( 10), H1472-H1480. doi:10.1152/ajpheart.00520.2013 -
NLM
Cunha NV da, Pinge-Filho P, Panis C, Silva BR, Pernomian L, Grando MD, Cecchini R, Bendhack LM, Martins-Pinge MC. Decreased endothelial nitric oxide, systemic oxidative stress, and increased sympathetic modulation contribute to hypertension in obese rats [Internet]. American Journal of Physiology Heart and Circulatory Physiology. 2014 ; 306( 10): H1472-H1480.[citado 2024 abr. 20 ] Available from: https://doi.org/10.1152/ajpheart.00520.2013 -
Vancouver
Cunha NV da, Pinge-Filho P, Panis C, Silva BR, Pernomian L, Grando MD, Cecchini R, Bendhack LM, Martins-Pinge MC. Decreased endothelial nitric oxide, systemic oxidative stress, and increased sympathetic modulation contribute to hypertension in obese rats [Internet]. American Journal of Physiology Heart and Circulatory Physiology. 2014 ; 306( 10): H1472-H1480.[citado 2024 abr. 20 ] Available from: https://doi.org/10.1152/ajpheart.00520.2013 - Endothelial cyclooxygenase differently modulates the contractile response to phenylephrine but not to thromboxane analog in aorta from normotensive and hypertensive rats
- Increased resting tension abolishes the endothelial anti-contractile effect induced by phenylephrine in renal hypertensive (2K-1C) rat aorta
- Hidrogen peroxide decreases the contractile response induced by phenylephrine in renal hypertensive rat aorta
- Augmented nitric oxide production and up-regulation of endothelial nitric oxide synthase during cecal ligation and perforation
- A tolerância induzida pela nitroglicerina pode ter início no endotélio após 5 minutos de exposição in vitro com aumento de EROs
- Relaxation induced by the nitric oxide donor and cyclooxygenase inhibitor NCX2121 in renal hypertensive rat aortas
- Endothelial NO-synthase is involved in decreased norepinephrine-induced contraction in 2K-1C rat aorta by alpha-adrenoceptor activation
- Inibição da NO-sintase normaliza a resposta contrátil estimulada com fenilefrina em aorta de ratos hipertensos renais 2R-1C
- Fenilefrina ativa a enzima NO-sintase em aorta de ratos heipertensos via AKT e fosforilação da serina1177
- Hydrogen peroxide modulates the contractile response induced by phenylephrine in renal hypertensive rat aorta
Informações sobre o DOI: 10.1152/ajpheart.00520.2013 (Fonte: oaDOI API)
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