Inflammatory aspects related to cardiac hypertrophy induced BY T3 (2014)
- Authors:
- USP affiliated authors: MORISCOT, ANSELMO SIGARI - ICB ; CHAVES, MARIA LUIZA MORAIS BARRETO DE - ICB
- Unidade: ICB
- Subjects: ANATOMIA; INFLAMAÇÃO; RATOS WISTAR; CITOCINAS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Resumos
- Conference titles: Reunião Anual da Federação de Sociedades de Biologia Experimental (FeSBE)
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ABNT
TAKANO, Ana Paula Cremasco e MORISCOT, Anselmo Sigari e BARRETO-CHAVES, Maria Luiza Morais. Inflammatory aspects related to cardiac hypertrophy induced BY T3. 2014, Anais.. Caxambu: FeSBE, 2014. . Acesso em: 18 abr. 2024. -
APA
Takano, A. P. C., Moriscot, A. S., & Barreto-Chaves, M. L. M. (2014). Inflammatory aspects related to cardiac hypertrophy induced BY T3. In Resumos. Caxambu: FeSBE. -
NLM
Takano APC, Moriscot AS, Barreto-Chaves MLM. Inflammatory aspects related to cardiac hypertrophy induced BY T3. Resumos. 2014 ;[citado 2024 abr. 18 ] -
Vancouver
Takano APC, Moriscot AS, Barreto-Chaves MLM. Inflammatory aspects related to cardiac hypertrophy induced BY T3. Resumos. 2014 ;[citado 2024 abr. 18 ] - Thyroid hormone increases beta-catenin levels through PI3K in rat heart in vivo and in vitro
- Thyroid hormone increases beta-catenin levels through PI3K in rat heart in vivo and in vitro
- Thyroid hormone down regulates myomesin gene expression in rat heart
- Efeito do hipertiroidismo experimental na expressão de calcineurina em tecidos cardíacos de ratos
- The activation of calcineurin pathway contributes to the cardiomyocyte hypertrophy induced by thyroid hormone
- M-Protein is down-regulated in cardiac hypertrophy driven by thyroid hormone in rats
- S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone
- Thyroid hormone down regulates M-protein expression in rat heart
- Effect creatine supplementation upon skeletal muscle wasting during immobilazation
- Interação do fator músculo específico mef-2a com o receptor do hormônio tiroideano na ativação do gene da serca-2
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