Calorie restriction remodels mitochondrial membranes by modulating the biosynthesis of cardiolipin (2015)
- Authors:
- Autor USP: KOWALTOWSKI, ALÍCIA JULIANA - IQ
- Unidade: IQ
- Subjects: FOSFOLIPÍDEOS; MITOCÔNDRIAS
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq)
- Publisher place: São Paulo
- Date published: 2015
- Source:
- Título do periódico: Abstracts Book
- Conference titles: Congress of the International Union for Biochemistry and Molecular Biology - IUBMB
-
ABNT
LUÉVANO-MARTÍNEZ, L. A et al. Calorie restriction remodels mitochondrial membranes by modulating the biosynthesis of cardiolipin. 2015, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq), 2015. . Acesso em: 20 abr. 2024. -
APA
Luévano-Martínez, L. A., Forni, M. F., Peloggia, J., & Kowaltowski, A. J. (2015). Calorie restriction remodels mitochondrial membranes by modulating the biosynthesis of cardiolipin. In Abstracts Book. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq). -
NLM
Luévano-Martínez LA, Forni MF, Peloggia J, Kowaltowski AJ. Calorie restriction remodels mitochondrial membranes by modulating the biosynthesis of cardiolipin. Abstracts Book. 2015 ;[citado 2024 abr. 20 ] -
Vancouver
Luévano-Martínez LA, Forni MF, Peloggia J, Kowaltowski AJ. Calorie restriction remodels mitochondrial membranes by modulating the biosynthesis of cardiolipin. Abstracts Book. 2015 ;[citado 2024 abr. 20 ] - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
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