Caspase-1 but not caspase-11 is required for NLRC4-mediated pyroptosis and restriction of infection by flagellated Legionella species in mouse macrophages and in vivo (2015)
- Authors:
- Autor USP: ZAMBONI, DARIO SIMÕES - FMRP
- Unidade: FMRP
- DOI: 10.4049/jimmunol.1501223
- Subjects: BACTÉRIAS GRAM-NEGATIVAS; PNEUMONIA; MACRÓFAGOS; PROTEÍNAS DE LIGAÇÃO DO CÁLCIO; APOPTOSE
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: The Journal of Immunology
- ISSN: 1550-6606
- Volume/Número/Paginação/Ano: v. 195, n. 5, p. 2303-2311, 2015
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: bronze
-
ABNT
CERQUEIRA, Daiane M. et al. Caspase-1 but not caspase-11 is required for NLRC4-mediated pyroptosis and restriction of infection by flagellated Legionella species in mouse macrophages and in vivo. The Journal of Immunology, v. 195, n. 5, p. 2303-2311, 2015Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1501223. Acesso em: 25 abr. 2024. -
APA
Cerqueira, D. M., Pereira, M. S. F., Silva, A. L. N., Cunha, L. D., & Zamboni, D. S. (2015). Caspase-1 but not caspase-11 is required for NLRC4-mediated pyroptosis and restriction of infection by flagellated Legionella species in mouse macrophages and in vivo. The Journal of Immunology, 195( 5), 2303-2311. doi:10.4049/jimmunol.1501223 -
NLM
Cerqueira DM, Pereira MSF, Silva ALN, Cunha LD, Zamboni DS. Caspase-1 but not caspase-11 is required for NLRC4-mediated pyroptosis and restriction of infection by flagellated Legionella species in mouse macrophages and in vivo [Internet]. The Journal of Immunology. 2015 ; 195( 5): 2303-2311.[citado 2024 abr. 25 ] Available from: https://doi.org/10.4049/jimmunol.1501223 -
Vancouver
Cerqueira DM, Pereira MSF, Silva ALN, Cunha LD, Zamboni DS. Caspase-1 but not caspase-11 is required for NLRC4-mediated pyroptosis and restriction of infection by flagellated Legionella species in mouse macrophages and in vivo [Internet]. The Journal of Immunology. 2015 ; 195( 5): 2303-2311.[citado 2024 abr. 25 ] Available from: https://doi.org/10.4049/jimmunol.1501223 - Role of inflammasomes in diffuse and localized cutaneous Leishmaniasis
- Autophagy downstream of endosomal Toll-like receptor signaling in macrophages is a key mechanism for resistance to Leishmania major infection
- Role of the transcriptional regulator SP140 in resistance to bacterial infections via repression of type I interferons
- Unique role of the Nlrc4 inflammasome for pore formation and pyroptosis in response to flagellated bacterial pathogens
- Nod-like receptors-dependent innate immune responses to intracellular pathogens
- NALP3: a key player in caspase-1 activation
- NOD1 and NOD2 signaling in infection and inflammation
- Participação dos receptores de reconhecimento padrão NOD1 e NOD2 no recrutamento de neutrófilos para o pulmão em resposta a infecção murina por legionella penumophila
- Critical role of ASC inflammasomes and bacterial type IV secretion system in caspase-1 activation and host innate resistance to Brucella abortus infection
- Inflammasome mediated innate immune responses to Leishmania
Informações sobre o DOI: 10.4049/jimmunol.1501223 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
