Dietary interesterified fat enriched with palmitic acid induces atherosclerosis by impairing macrophage cholesterol efflux and eliciting inflammation (2016)
- Authors:
- Afonso, Milessa Silva
- Lavrador, Maria Silvia Ferrari
- Koike, Márcia Kiyomi
- Cintra, Dennys Esper - Universidade Estadual de Campinas (UNICAMP)
- Ferreira, Fabiana Dias
- Nunes, Valéria Sutti
- Castilho, Gabriela
- Gioielli, Luiz Antonio
- Bombo, Renata Paula
- Catanozi, Sérgio
- Caldini, Elia Garcia
- Rodrigues, Nilsa Regina Damasceno
- Passarelli, Marisa
- Nakandakare, Edna Regina
- Lottenberg, Ana Maria
- USP affiliated authors: KOIKE, MARCIA KIYOMI - FM ; GIOIELLI, LUIZ ANTONIO - FCF ; CATANOZI, SERGIO - FM ; PASSARELLI, MARISA - FM ; NAKANDAKARE, EDNA REGINA - FM
- Unidades: FM; FCF
- DOI: 10.1016/j.jnutbio.2016.01.005
- Subjects: ARTERIOSCLEROSE; INFLAMAÇÃO; DIETA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Journal of Nutritional Biochemistry
- ISSN: 0955-2863
- Volume/Número/Paginação/Ano: v. 32, p. 91-100, 2016
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
AFONSO, Milessa Silva et al. Dietary interesterified fat enriched with palmitic acid induces atherosclerosis by impairing macrophage cholesterol efflux and eliciting inflammation. Journal of Nutritional Biochemistry, v. 32, p. 91-100, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.jnutbio.2016.01.005. Acesso em: 23 abr. 2024. -
APA
Afonso, M. S., Lavrador, M. S. F., Koike, M. K., Cintra, D. E., Ferreira, F. D., Nunes, V. S., et al. (2016). Dietary interesterified fat enriched with palmitic acid induces atherosclerosis by impairing macrophage cholesterol efflux and eliciting inflammation. Journal of Nutritional Biochemistry, 32, 91-100. doi:10.1016/j.jnutbio.2016.01.005 -
NLM
Afonso MS, Lavrador MSF, Koike MK, Cintra DE, Ferreira FD, Nunes VS, Castilho G, Gioielli LA, Bombo RP, Catanozi S, Caldini EG, Rodrigues NRD, Passarelli M, Nakandakare ER, Lottenberg AM. Dietary interesterified fat enriched with palmitic acid induces atherosclerosis by impairing macrophage cholesterol efflux and eliciting inflammation [Internet]. Journal of Nutritional Biochemistry. 2016 ; 32 91-100.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.jnutbio.2016.01.005 -
Vancouver
Afonso MS, Lavrador MSF, Koike MK, Cintra DE, Ferreira FD, Nunes VS, Castilho G, Gioielli LA, Bombo RP, Catanozi S, Caldini EG, Rodrigues NRD, Passarelli M, Nakandakare ER, Lottenberg AM. Dietary interesterified fat enriched with palmitic acid induces atherosclerosis by impairing macrophage cholesterol efflux and eliciting inflammation [Internet]. Journal of Nutritional Biochemistry. 2016 ; 32 91-100.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.jnutbio.2016.01.005 - Interesterifi ed Fats Induce Liver Neutrophils Infi ltration, Fibrosis, and JNK activation
- A dietary trans fat substitute also induces atherosclerosis development in ldlr-ko mice
- Exercise Training Favorably Modulates Gene and Protein Expression That Regulate Arterial Cholesterol Content in CETP Transgenic Mice
- Advanced glycated albumin primers macrophages to S100B calgranulins and LPS stimulation that impairs the macrophage reverse cholesterol transport
- The reverse cholesterol transport system is not influenced by the HDL composition modification or the reduced macrophage ABCG-1 expression in chronic kidney disease rats
- Interesterified fats induce deleterious effects on adipose tissue and liver in LDLr-KO mice
- Aerobic Exercise Training Selectively Changes Oxysterol Levels and Metabolism Reducing Cholesterol Accumulation in the Aorta of Dyslipidemic Mice
- N-acetylcysteine prevents endoplasmic reticulum stress elicited in macrophages by serum albumin drawn from chronic kidney disease rats and selectively affects lipid transporters, ABCA-1 and ABCG-1
- Endoplasmic reticulum stress inhibition prevents ABCA-1 reductio in glycated albumin-treated macrophages
- Obesidade e metabolismo de lipídios
Informações sobre o DOI: 10.1016/j.jnutbio.2016.01.005 (Fonte: oaDOI API)
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