IKKβ is a potential anti-angiogenic therapeutic target in KRAS-induced lung cancer (2016)
- Autor:
- Autor USP: BASSÈRES, DANIELA SANCHEZ - IQ
- Unidade: IQ
- Assunto: NEOPLASIAS PULMONARES
- Language: Inglês
- Imprenta:
- Publisher: American Association for Cancer Research
- Publisher place: Philadelphia
- Date published: 2016
- Source:
- Título do periódico: Abstracts
- Conference titles: Precision Medicine Series: Targeting the Vulnerabilities of Cancer
-
ABNT
BASSÈRES, Daniela Sanchez. IKKβ is a potential anti-angiogenic therapeutic target in KRAS-induced lung cancer. 2016, Anais.. Philadelphia: American Association for Cancer Research, 2016. . Acesso em: 23 abr. 2024. -
APA
Bassères, D. S. (2016). IKKβ is a potential anti-angiogenic therapeutic target in KRAS-induced lung cancer. In Abstracts. Philadelphia: American Association for Cancer Research. -
NLM
Bassères DS. IKKβ is a potential anti-angiogenic therapeutic target in KRAS-induced lung cancer. Abstracts. 2016 ;[citado 2024 abr. 23 ] -
Vancouver
Bassères DS. IKKβ is a potential anti-angiogenic therapeutic target in KRAS-induced lung cancer. Abstracts. 2016 ;[citado 2024 abr. 23 ] - Aurora kinases: potential therapeutic targets in K-Ras-induced lung cancer
- Targeted BMI1 inhibition impairs tumor growth in lung adenocarcinomas with low CEBP alpha expression
- Exploring IKKβ kinase as a therapeutic target for KRAS-driven lung tumor initiating cells
- Exploring IKKβ as an anti-metastatic therapeutic target in KRAS-induced lung cancer
- Targeting Aurora A kinase activity in lung cancer initiating cells driven by oncogenic KRAS
- IKKβ inhibition suppresses sphere formation and self renewal of lung cancer initiating cells
- Aurora kinase targeting in lung cancer reduces KRAS-induced transformation
- IKKβ kinase promotes stemness, migration, and invasion in KRAS-driven lung adenocarcinoma cells
- Identification of a targetable KRAS-mutant epithelial population in non-small cell lung cancer
- FOXO1 inhibition by oncogenic KRAS as an important mechanism promoting lung oncogenesis
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