Diazoxide prevents reactive oxygen species and mitochondrial damage, leading to anti-hypertrophic effects (2017)
- Authors:
- Autor USP: KOWALTOWSKI, ALÍCIA JULIANA - IQ
- Unidade: IQ
- DOI: 10.1016/j.cbi.2016.11.012
- Subjects: MITOCÔNDRIAS; ESTRESSE OXIDATIVO; RADICAIS LIVRES; CÁLCIO
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Chemico Biological Interactions
- ISSN: 0009-2797
- Volume/Número/Paginação/Ano: v. 261, p. 50-55, 2017
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
LUCAS, Aline M et al. Diazoxide prevents reactive oxygen species and mitochondrial damage, leading to anti-hypertrophic effects. Chemico Biological Interactions, v. 261, p. 50-55, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2016.11.012. Acesso em: 19 abr. 2024. -
APA
Lucas, A. M., Caldas, F. R., Silva, A. P. da, Ventura, M. M., Leite, I. M., Filgueiras, A. B., et al. (2017). Diazoxide prevents reactive oxygen species and mitochondrial damage, leading to anti-hypertrophic effects. Chemico Biological Interactions, 261, 50-55. doi:10.1016/j.cbi.2016.11.012 -
NLM
Lucas AM, Caldas FR, Silva AP da, Ventura MM, Leite IM, Filgueiras AB, Silva CGL, Kowaltowski AJ, Facundo HT. Diazoxide prevents reactive oxygen species and mitochondrial damage, leading to anti-hypertrophic effects [Internet]. Chemico Biological Interactions. 2017 ; 261 50-55.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.cbi.2016.11.012 -
Vancouver
Lucas AM, Caldas FR, Silva AP da, Ventura MM, Leite IM, Filgueiras AB, Silva CGL, Kowaltowski AJ, Facundo HT. Diazoxide prevents reactive oxygen species and mitochondrial damage, leading to anti-hypertrophic effects [Internet]. Chemico Biological Interactions. 2017 ; 261 50-55.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.cbi.2016.11.012 - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
Informações sobre o DOI: 10.1016/j.cbi.2016.11.012 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
