Lack of XPC leads to a shift between respiratory complexes I and II but sensitizes cells to mitochondrial stress (2017)
- Authors:
- Mori, Mateus Prates
- Costa, Rute Alves Pereira e - Universidade Estadual de Campinas (UNICAMP)
- Soltys, Daniela Tathiana
- Freire, Thiago de Souza
- Rossato, Franco Aparecido - Universidade Estadual de Campinas (UNICAMP)
- Amigo, Ignácio
- Kowaltowski, Alicia Juliana
- Vercesi, Anibal Eugenio - Universidade Estadual de Campinas (UNICAMP)
- Souza-Pinto, Nadja Cristhina de
- USP affiliated authors: KOWALTOWSKI, ALÍCIA JULIANA - IQ ; PINTO, NADJA CRISTHINA DE SOUZA - IQ
- Unidade: IQ
- DOI: 10.1038/s41598-017-00130-x
- Subjects: MITOCÔNDRIAS; GENÔMICA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Scientific Reports
- ISSN: 2045-2322
- Volume/Número/Paginação/Ano: v. 7, p. 1-15 art. 155, 2017
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
MORI, Mateus Prates et al. Lack of XPC leads to a shift between respiratory complexes I and II but sensitizes cells to mitochondrial stress. Scientific Reports, v. 7, p. 1-15 art. 155, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-00130-x. Acesso em: 25 abr. 2024. -
APA
Mori, M. P., Costa, R. A. P. e, Soltys, D. T., Freire, T. de S., Rossato, F. A., Amigo, I., et al. (2017). Lack of XPC leads to a shift between respiratory complexes I and II but sensitizes cells to mitochondrial stress. Scientific Reports, 7, 1-15 art. 155. doi:10.1038/s41598-017-00130-x -
NLM
Mori MP, Costa RAP e, Soltys DT, Freire T de S, Rossato FA, Amigo I, Kowaltowski AJ, Vercesi AE, Souza-Pinto NC de. Lack of XPC leads to a shift between respiratory complexes I and II but sensitizes cells to mitochondrial stress [Internet]. Scientific Reports. 2017 ; 7 1-15 art. 155.[citado 2024 abr. 25 ] Available from: https://doi.org/10.1038/s41598-017-00130-x -
Vancouver
Mori MP, Costa RAP e, Soltys DT, Freire T de S, Rossato FA, Amigo I, Kowaltowski AJ, Vercesi AE, Souza-Pinto NC de. Lack of XPC leads to a shift between respiratory complexes I and II but sensitizes cells to mitochondrial stress [Internet]. Scientific Reports. 2017 ; 7 1-15 art. 155.[citado 2024 abr. 25 ] Available from: https://doi.org/10.1038/s41598-017-00130-x - Mitochondria and reactive oxygen species
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- The mitochondrial electron transport chain is necessary for life span extension mediated by calorie restriction in Saccharomyces cerevisiae
- mtDNA and electron transport chain integrity in lifespan extention mediated by calorie restriction in Saccharomyces cerevisiae
- Removal of oxidative DNA damage via FEN1-dependent long-patch base excision repair in human cell mitochondria
- Cockayne syndrome group B protein promotes mitochondrial DNA stability by supporting the DNA repair association with the mitochondrial membrane
- Characterization of oxidative guanine damage and repair in mammalian telomeres
- The role of mitochondrial DNA damage in the citotoxicity of reactive oxygen species
- Formation and repair of oxidative damage in the mitochondrial DNA
Informações sobre o DOI: 10.1038/s41598-017-00130-x (Fonte: oaDOI API)
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