Cell Culture Models of Fatty Acid Overload: Problems and Solutions (2018)
- Authors:
- Autor USP: KOWALTOWSKI, ALÍCIA JULIANA - IQ
- Unidade: IQ
- DOI: 10.1016/j.bbalip.2017.11.006
- Subjects: ÁCIDOS GRAXOS; LIPÍDEOS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Biochimica et Biophysica Acta
- ISSN: 1388-1981
- Volume/Número/Paginação/Ano: v. 1863, n. 2, p. 143-151, 2018
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: hybrid
- Licença: publisher-specific-oa
-
ABNT
ALSABEEH, Nour et al. Cell Culture Models of Fatty Acid Overload: Problems and Solutions. Biochimica et Biophysica Acta, v. 1863, n. 2, p. 143-151, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.bbalip.2017.11.006. Acesso em: 14 maio 2024. -
APA
Alsabeeh, N., Chausse, B., Kakimoto, P. A., Kowaltowski, A. J., & Shirihai, O. (2018). Cell Culture Models of Fatty Acid Overload: Problems and Solutions. Biochimica et Biophysica Acta, 1863( 2), 143-151. doi:10.1016/j.bbalip.2017.11.006 -
NLM
Alsabeeh N, Chausse B, Kakimoto PA, Kowaltowski AJ, Shirihai O. Cell Culture Models of Fatty Acid Overload: Problems and Solutions [Internet]. Biochimica et Biophysica Acta. 2018 ; 1863( 2): 143-151.[citado 2024 maio 14 ] Available from: https://doi.org/10.1016/j.bbalip.2017.11.006 -
Vancouver
Alsabeeh N, Chausse B, Kakimoto PA, Kowaltowski AJ, Shirihai O. Cell Culture Models of Fatty Acid Overload: Problems and Solutions [Internet]. Biochimica et Biophysica Acta. 2018 ; 1863( 2): 143-151.[citado 2024 maio 14 ] Available from: https://doi.org/10.1016/j.bbalip.2017.11.006 - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
Informações sobre o DOI: 10.1016/j.bbalip.2017.11.006 (Fonte: oaDOI API)
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