Epigenetic mechanisms in liver tumors: gene expression analysis of epigenetic machinery in hepatoblastomas (2018)
- Authors:
- USP affiliated authors: ROSENBERG, CARLA - IB ; ZATZ, MAYANA - IB ; KREPISCHI, ANA CRISTINA VICTORINO - IB
- Unidade: IB
- DOI: 10.1158/1557-3265.TCM17-A10
- Subjects: NEOPLASIAS HEPÁTICAS; CARCINOGÊNESE; EPIGÊNESE GENÉTICA; EXPRESSÃO GÊNICA; CRIANÇAS
- Language: Inglês
- Imprenta:
- Publisher place: Philadelphia
- Date published: 2018
- Source:
- Título do periódico: Clinical Cancer Research
- ISSN: 1078-0432
- Volume/Número/Paginação/Ano: v. 24, n. 1, suppl., res. A10, 2018
- Conference titles: AACR International Conference on Translational Cancer Medicine
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
RIVAS, Maria et al. Epigenetic mechanisms in liver tumors: gene expression analysis of epigenetic machinery in hepatoblastomas. Clinical Cancer Research. Philadelphia: Instituto de Biociências, Universidade de São Paulo. Disponível em: https://doi.org/10.1158/1557-3265.TCM17-A10. Acesso em: 18 abr. 2024. , 2018 -
APA
Rivas, M., Costa, C. da, Toledo, S., Cypriano, M., Caires, L., Goulart, E., et al. (2018). Epigenetic mechanisms in liver tumors: gene expression analysis of epigenetic machinery in hepatoblastomas. Clinical Cancer Research. Philadelphia: Instituto de Biociências, Universidade de São Paulo. doi:10.1158/1557-3265.TCM17-A10 -
NLM
Rivas M, Costa C da, Toledo S, Cypriano M, Caires L, Goulart E, Carraro D, Werneck I, Rosenberg C, Zatz M, Krepischi ACV. Epigenetic mechanisms in liver tumors: gene expression analysis of epigenetic machinery in hepatoblastomas [Internet]. Clinical Cancer Research. 2018 ; 24( 1):[citado 2024 abr. 18 ] Available from: https://doi.org/10.1158/1557-3265.TCM17-A10 -
Vancouver
Rivas M, Costa C da, Toledo S, Cypriano M, Caires L, Goulart E, Carraro D, Werneck I, Rosenberg C, Zatz M, Krepischi ACV. Epigenetic mechanisms in liver tumors: gene expression analysis of epigenetic machinery in hepatoblastomas [Internet]. Clinical Cancer Research. 2018 ; 24( 1):[citado 2024 abr. 18 ] Available from: https://doi.org/10.1158/1557-3265.TCM17-A10 - Cytogenetically visible inversions are formed by multiple molecular mechanisms
- LINE-1 hypermethylation in peripheral blood of cutaneous melanoma patients is associated with metastasis
- Rare germline copy number variations in hereditary cutaneous melanoma
- Lymphovascular invasion and histologic grade are associated with specific genomic profiles in invasive carcinomas of the breast
- Genetic testing for idiopathic intellectual disability: use of DNA extracted from saliva for SNP Genotyping
- Does increase in genomic microarray resolution result in increased diagnostic yield?
- Insights into the somatic mutation burden of hepatoblastomas using whole exome sequencing
- Genotype–phenotype correlation of 16p13.3 terminal duplication and 22q13.33 deletion: natural history of a patient and review of the literature
- Large germline copy number variations as predisposing factor in childhood neoplasms
- The profile and contribution of rare germline copy number variants to cancer risk in Li-Fraumeni patients negative for TP53 mutations
Informações sobre o DOI: 10.1158/1557-3265.TCM17-A10 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas