Obesity induces artery-specific alterations: evaluation of vascular function and inflammatory and smooth muscle phenotypic markers (2017)
- Authors:
- USP affiliated authors: SOARES JÚNIOR, ANTONIO GARCIA - ICB ; CARVALHO, MARIA HELENA CATELLI DE - ICB ; AKAMINE, ELIANA HIROMI - ICB
- Unidade: ICB
- DOI: 10.1155/2017/5038602
- Assunto: FARMACOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: BioMed Research International
- ISSN: 2314-6141
- Volume/Número/Paginação/Ano: v. 2017, ID 5038602, p. 1-10, 2017
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
SOARES, Antonio Garcia e CARVALHO, Maria Helena Catelli de e AKAMINE, Eliana Hiromi. Obesity induces artery-specific alterations: evaluation of vascular function and inflammatory and smooth muscle phenotypic markers. BioMed Research International, v. 2017, p. 1-10, 2017Tradução . . Disponível em: https://doi.org/10.1155/2017/5038602. Acesso em: 19 abr. 2024. -
APA
Soares, A. G., Carvalho, M. H. C. de, & Akamine, E. H. (2017). Obesity induces artery-specific alterations: evaluation of vascular function and inflammatory and smooth muscle phenotypic markers. BioMed Research International, 2017, 1-10. doi:10.1155/2017/5038602 -
NLM
Soares AG, Carvalho MHC de, Akamine EH. Obesity induces artery-specific alterations: evaluation of vascular function and inflammatory and smooth muscle phenotypic markers [Internet]. BioMed Research International. 2017 ; 2017 1-10.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1155/2017/5038602 -
Vancouver
Soares AG, Carvalho MHC de, Akamine EH. Obesity induces artery-specific alterations: evaluation of vascular function and inflammatory and smooth muscle phenotypic markers [Internet]. BioMed Research International. 2017 ; 2017 1-10.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1155/2017/5038602 - Intrauterine growth restriction leads to a high-corticosterone producing offspring: an implication for pulmonary infection susceptibility
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Informações sobre o DOI: 10.1155/2017/5038602 (Fonte: oaDOI API)
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