Novel evidence that extracellular nucleotides and purinergic signaling induce innate immunity-mediated mobilization of hematopoietic stem/progenitor cells (2018)
- Authors:
- Autor USP: ULRICH, ALEXANDER HENNING - IQ
- Unidade: IQ
- DOI: 10.1038/s41375-018-0122-0
- Subjects: CÉLULAS-TRONCO; NUCLEOTÍDEOS
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: hybrid
- Licença: cc-by-nc-nd
-
ABNT
ADAMIAK, Mateusz et al. Novel evidence that extracellular nucleotides and purinergic signaling induce innate immunity-mediated mobilization of hematopoietic stem/progenitor cells. Leukemia, v. 32, p. 1920-1931, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41375-018-0122-0. Acesso em: 26 abr. 2024. -
APA
Adamiak, M., Bujko, K., Cymer, M., Plonka, M., Glaser, T., Kucia, M., et al. (2018). Novel evidence that extracellular nucleotides and purinergic signaling induce innate immunity-mediated mobilization of hematopoietic stem/progenitor cells. Leukemia, 32, 1920-1931. doi:10.1038/s41375-018-0122-0 -
NLM
Adamiak M, Bujko K, Cymer M, Plonka M, Glaser T, Kucia M, Ratajczak J, Ulrich H, Latif AA, Ratajczak MZ. Novel evidence that extracellular nucleotides and purinergic signaling induce innate immunity-mediated mobilization of hematopoietic stem/progenitor cells [Internet]. Leukemia. 2018 ; 32 1920-1931.[citado 2024 abr. 26 ] Available from: https://doi.org/10.1038/s41375-018-0122-0 -
Vancouver
Adamiak M, Bujko K, Cymer M, Plonka M, Glaser T, Kucia M, Ratajczak J, Ulrich H, Latif AA, Ratajczak MZ. Novel evidence that extracellular nucleotides and purinergic signaling induce innate immunity-mediated mobilization of hematopoietic stem/progenitor cells [Internet]. Leukemia. 2018 ; 32 1920-1931.[citado 2024 abr. 26 ] Available from: https://doi.org/10.1038/s41375-018-0122-0 - Aluminum-induced alterations of purinergic signalling in embryonic neural progenitor cells
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Informações sobre o DOI: 10.1038/s41375-018-0122-0 (Fonte: oaDOI API)
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