Resilient hepatic mitochondrial function in diet-induced liver inflammation and insulin resistance (2018)
- Authors:
- USP affiliated authors: ORTIZ, CAMILLE CRISTINE CALDEIRA - IQ ; KOWALTOWSKI, ALÍCIA JULIANA - IQ
- Unidade: IQ
- Subjects: OBESIDADE; MITOCÔNDRIAS
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Bioquímica e Biologia Molecular/SBBq
- Publisher place: São Paulo
- Date published: 2018
- Source:
- Título do periódico: Abstracts
- Conference titles: Annual Meeting of the Brazilian Society for Biochemistry and Molecular Biology/SBBq
-
ABNT
KAKIMOTO, Pâmela Aiako Hypólito Brito et al. Resilient hepatic mitochondrial function in diet-induced liver inflammation and insulin resistance. 2018, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular/SBBq, 2018. Disponível em: http://www.sbbq.org.br/reuniao/2018/livro_resumos_2018.pdf. Acesso em: 19 abr. 2024. -
APA
Kakimoto, P. A. H. B., Freitas, B. C. de, Ortiz, C. C. C., & Kowaltowski, A. J. (2018). Resilient hepatic mitochondrial function in diet-induced liver inflammation and insulin resistance. In Abstracts. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular/SBBq. Recuperado de http://www.sbbq.org.br/reuniao/2018/livro_resumos_2018.pdf -
NLM
Kakimoto PAHB, Freitas BC de, Ortiz CCC, Kowaltowski AJ. Resilient hepatic mitochondrial function in diet-induced liver inflammation and insulin resistance [Internet]. Abstracts. 2018 ;[citado 2024 abr. 19 ] Available from: http://www.sbbq.org.br/reuniao/2018/livro_resumos_2018.pdf -
Vancouver
Kakimoto PAHB, Freitas BC de, Ortiz CCC, Kowaltowski AJ. Resilient hepatic mitochondrial function in diet-induced liver inflammation and insulin resistance [Internet]. Abstracts. 2018 ;[citado 2024 abr. 19 ] Available from: http://www.sbbq.org.br/reuniao/2018/livro_resumos_2018.pdf - Resilient hepatic mitochondrial function and lack of iNOS dependence in diet-induced insulin resistance
- Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
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