Autophagy downstream of endosomal Toll-like receptor signaling in macrophages is a key mechanism for resistance to Leishmania major infection (2017)
- Authors:
- Autor USP: ZAMBONI, DARIO SIMÕES - FMRP
- Unidade: FMRP
- DOI: 10.1074/jbc.m117.780981
- Subjects: LEISHMANIA; MACRÓFAGOS
- Keywords: AUTOPHAGY; INFECTION; LEISHMANIA; MACROPHAGE; TOLL-LIKE RECEPTOR (TLR)
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Journal of Biological Chemistry
- ISSN: 0021-9258
- Volume/Número/Paginação/Ano: v. 292, n. 32, p. 13087-13096, 2017
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: hybrid
- Licença: cc-by
-
ABNT
FRANCO, Luís Henrique et al. Autophagy downstream of endosomal Toll-like receptor signaling in macrophages is a key mechanism for resistance to Leishmania major infection. Journal of Biological Chemistry, v. 292, n. 32, p. 13087-13096, 2017Tradução . . Disponível em: https://doi.org/10.1074/jbc.m117.780981. Acesso em: 18 abr. 2024. -
APA
Franco, L. H., Fleuri, A. K. A., Pellison, N. C., Quirino, G. F. da S., Horta, C. V., Carvalho, R. V. H. de, et al. (2017). Autophagy downstream of endosomal Toll-like receptor signaling in macrophages is a key mechanism for resistance to Leishmania major infection. Journal of Biological Chemistry, 292( 32), 13087-13096. doi:10.1074/jbc.m117.780981 -
NLM
Franco LH, Fleuri AKA, Pellison NC, Quirino GF da S, Horta CV, Carvalho RVH de, Oliveira SC, Zamboni DS. Autophagy downstream of endosomal Toll-like receptor signaling in macrophages is a key mechanism for resistance to Leishmania major infection [Internet]. Journal of Biological Chemistry. 2017 ; 292( 32): 13087-13096.[citado 2024 abr. 18 ] Available from: https://doi.org/10.1074/jbc.m117.780981 -
Vancouver
Franco LH, Fleuri AKA, Pellison NC, Quirino GF da S, Horta CV, Carvalho RVH de, Oliveira SC, Zamboni DS. Autophagy downstream of endosomal Toll-like receptor signaling in macrophages is a key mechanism for resistance to Leishmania major infection [Internet]. Journal of Biological Chemistry. 2017 ; 292( 32): 13087-13096.[citado 2024 abr. 18 ] Available from: https://doi.org/10.1074/jbc.m117.780981 - Role of inflammasomes in diffuse and localized cutaneous Leishmaniasis
- Role of the transcriptional regulator SP140 in resistance to bacterial infections via repression of type I interferons
- Unique role of the Nlrc4 inflammasome for pore formation and pyroptosis in response to flagellated bacterial pathogens
- Nod-like receptors-dependent innate immune responses to intracellular pathogens
- NALP3: a key player in caspase-1 activation
- NOD1 and NOD2 signaling in infection and inflammation
- Participação dos receptores de reconhecimento padrão NOD1 e NOD2 no recrutamento de neutrófilos para o pulmão em resposta a infecção murina por legionella penumophila
- Critical role of ASC inflammasomes and bacterial type IV secretion system in caspase-1 activation and host innate resistance to Brucella abortus infection
- Inflammasome mediated innate immune responses to Leishmania
- Inhibition of non-canonical inflammasome activation in response to Legionella pneumophila by a Dot/Icm effector of Coxiella burnetii
Informações sobre o DOI: 10.1074/jbc.m117.780981 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
