Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state (2018)
- Authors:
- Autor USP: KOWALTOWSKI, ALÍCIA JULIANA - IQ
- Unidade: IQ
- Subjects: ANTIOXIDANTES; ESTRESSE OXIDATIVO; MITOCÔNDRIAS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Resumos
- Conference titles: Reunião Anual da Federação de Sociedades de Biologia Experimental/FeSBE
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ABNT
DAVID, Cicera Edna Barbosa et al. Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state. 2018, Anais.. São Paulo: FeSBE, 2018. . Acesso em: 17 abr. 2024. -
APA
David, C. E. B., Lucas, A. M. B., Araújo, M. T. S., Coelho, B. N., Soares Neto, J. B., Portela, B. R. C., et al. (2018). Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state. In Resumos. São Paulo: FeSBE. -
NLM
David CEB, Lucas AMB, Araújo MTS, Coelho BN, Soares Neto JB, Portela BRC, Varela ALN, Kowaltowski AJ, Facundo HDTF. Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state. Resumos. 2018 ;[citado 2024 abr. 17 ] -
Vancouver
David CEB, Lucas AMB, Araújo MTS, Coelho BN, Soares Neto JB, Portela BRC, Varela ALN, Kowaltowski AJ, Facundo HDTF. Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state. Resumos. 2018 ;[citado 2024 abr. 17 ] - Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
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- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
- Efeito da morfologia mitocondrial sobre a capacidade de retenção de cálcio mitocondrial
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