Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection (2017)
- Authors:
- USP affiliated authors: CUNHA, FERNANDO DE QUEIROZ - FMRP ; SILVA, JOÃO SANTANA DA - FMRP ; PEREIRA, VANESSA CARREGARO - FMRP
- Unidade: FMRP
- DOI: 10.3389/fmicb.2017.00262
- Subjects: LEISHMANIA INFANTUM; CÉLULAS DENDRÍTICAS; LEISHMANIOSE VISCERAL; NEUTRÓFILOS
- Keywords: TLR2; Dendritic cell maturation; Neutrophil activation; Visceral leishmaniasis
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Frontiers in Microbiology
- ISSN: 1664-302X
- Volume/Número/Paginação/Ano: v. 8, art. 262, 2017
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
SACRAMENTO, Laís A. et al. Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection. Frontiers in Microbiology, v. 8, 2017Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2017.00262. Acesso em: 19 abr. 2024. -
APA
Sacramento, L. A., Costa, J. L. da, Lima, M. H. F. de, Sampaio, P. A., Almeida, R. P., Cunha, F. Q., et al. (2017). Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection. Frontiers in Microbiology, 8. doi:10.3389/fmicb.2017.00262 -
NLM
Sacramento LA, Costa JL da, Lima MHF de, Sampaio PA, Almeida RP, Cunha FQ, Silva JS da, Carregaro V. Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection [Internet]. Frontiers in Microbiology. 2017 ; 8[citado 2024 abr. 19 ] Available from: https://doi.org/10.3389/fmicb.2017.00262 -
Vancouver
Sacramento LA, Costa JL da, Lima MHF de, Sampaio PA, Almeida RP, Cunha FQ, Silva JS da, Carregaro V. Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection [Internet]. Frontiers in Microbiology. 2017 ; 8[citado 2024 abr. 19 ] Available from: https://doi.org/10.3389/fmicb.2017.00262 - Dual effect of Lutzomyia longipalpis saliva on Leishmania braziliensis infection is mediated by distinct saliva-induced
- Leishmania infantum Parasites subvert the host inflammatory response through the adenosine A2A receptor to promote the establishment of infection
- Toll-like receptor 9 signaling in dendritic cells regulates neutrophil recruitment to inflammatory foci following Leishmania infantum infection
- Protective role of 5-lipoxigenase during Leishmania infantum infection is associated with TH17 subset
- Interleukin-27 (IL-27) mediates susceptibility to visceral Leishmaniasis by suppressing the IL-17-neutrophil response
- T1/ST2 deficient mice display protection against Leishmania infantum experimental infection
- CCR2 signaling contributes to the differentiation of protective inflammatory dendritic cells in Leishmania braziliensis infection
- Interleukin 17A acts synergistically with interferon γ to promote protection against Leishmania infantum infection
- T cell immunity to Zika virus targets immunodominant epitopes that show cross-reactivity with other Flaviviruses
- Nucleosides present on phlebotomine saliva induce immunossuppression and promote the infection establishment
Informações sobre o DOI: 10.3389/fmicb.2017.00262 (Fonte: oaDOI API)
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