Relaxation induced by the nitric oxide donor and cyclooxygenase inhibitor NCX2121 in renal hypertensive rat aortas (2017)
- Authors:
- USP affiliated authors: GRANDO, MARCELLA DARUGE - FCFRP ; BENDHACK, LUSIANE MARIA - FCFRP
- Unidade: FCFRP
- DOI: 10.1016/j.ejps.2017.06.007
- Subjects: AORTA TORÁCICA; ÓXIDO NÍTRICO; HIPERTENSÃO; VASODILATAÇÃO
- Keywords: Renovascular hypertension; Vasodilatation; Nitric oxide; Cyclooxygenase; NO-NSAID
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: European Journal of Pharmaceutical Sciences
- ISSN: 0928-0987
- Volume/Número/Paginação/Ano: v. 107, p. 45-53, 2017
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
PAULA, Tiago Dal-Cin de et al. Relaxation induced by the nitric oxide donor and cyclooxygenase inhibitor NCX2121 in renal hypertensive rat aortas. European Journal of Pharmaceutical Sciences, v. 107, p. 45-53, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.ejps.2017.06.007. Acesso em: 24 abr. 2024. -
APA
Paula, T. D. -C. de, Silva, B. R., Grando, M. D., Pernomian, L., Prado, A. F. do, & Bendhack, L. M. (2017). Relaxation induced by the nitric oxide donor and cyclooxygenase inhibitor NCX2121 in renal hypertensive rat aortas. European Journal of Pharmaceutical Sciences, 107, 45-53. doi:10.1016/j.ejps.2017.06.007 -
NLM
Paula TD-C de, Silva BR, Grando MD, Pernomian L, Prado AF do, Bendhack LM. Relaxation induced by the nitric oxide donor and cyclooxygenase inhibitor NCX2121 in renal hypertensive rat aortas [Internet]. European Journal of Pharmaceutical Sciences. 2017 ; 107 45-53.[citado 2024 abr. 24 ] Available from: https://doi.org/10.1016/j.ejps.2017.06.007 -
Vancouver
Paula TD-C de, Silva BR, Grando MD, Pernomian L, Prado AF do, Bendhack LM. Relaxation induced by the nitric oxide donor and cyclooxygenase inhibitor NCX2121 in renal hypertensive rat aortas [Internet]. European Journal of Pharmaceutical Sciences. 2017 ; 107 45-53.[citado 2024 abr. 24 ] Available from: https://doi.org/10.1016/j.ejps.2017.06.007 - Endothelial cyclooxygenase differently modulates the contractile response to phenylephrine but not to thromboxane analog in aorta from normotensive and hypertensive rats
- Decreased endothelial nitric oxide, systemic oxidative stress, and increased sympathetic modulation contribute to hypertension in obese rats
- Increased resting tension abolishes the endothelial anti-contractile effect induced by phenylephrine in renal hypertensive (2K-1C) rat aorta
- Hidrogen peroxide decreases the contractile response induced by phenylephrine in renal hypertensive rat aorta
- Augmented nitric oxide production and up-regulation of endothelial nitric oxide synthase during cecal ligation and perforation
- A tolerância induzida pela nitroglicerina pode ter início no endotélio após 5 minutos de exposição in vitro com aumento de EROs
- Endothelial NO-synthase is involved in decreased norepinephrine-induced contraction in 2K-1C rat aorta by alpha-adrenoceptor activation
- Contractile response induced by phenylephrine in modulated by eNOS phosphorylation and by hydrogen peroxide production in renal hypertensive rat aorta
- Tolerance to nitroglycerin is due to eNOS uncoupling
- Vascular activation of K+ channels and Na+-K+ ATPase activity of estrogen-deficient female rats
Informações sobre o DOI: 10.1016/j.ejps.2017.06.007 (Fonte: oaDOI API)
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