Prolactin protects against cytokine-induced beta-cell death by NFκB and JNK inhibition (2018)
- Authors:
- USP affiliated authors: FUJITA, ANDRÉ - IME ; ORTIS, FERNANDA - ICB
- Unidades: IME; ICB
- DOI: 10.1530/JME-16-0257
- Subjects: DIABETES MELLITUS; PROLACTINA; BIOINFORMÁTICA
- Keywords: NFkB; pancreatic beta cells; pro-inflammatory cytokines; STAT3
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Journal of Molecular Endocrinology
- ISSN: 0952-5041
- Volume/Número/Paginação/Ano: v. 61, n. 1, p. 25-36, 2018
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: bronze
-
ABNT
NARDELLI, Tarlliza Romanna et al. Prolactin protects against cytokine-induced beta-cell death by NFκB and JNK inhibition. Journal of Molecular Endocrinology, v. 61, n. 1, p. 25-36, 2018Tradução . . Disponível em: https://doi.org/10.1530/JME-16-0257. Acesso em: 23 abr. 2024. -
APA
Nardelli, T. R., Vanzela, E. C., Benedicto, K. C., Brozzi, F., Fujita, A., Cardozo, A. K., et al. (2018). Prolactin protects against cytokine-induced beta-cell death by NFκB and JNK inhibition. Journal of Molecular Endocrinology, 61( 1), 25-36. doi:10.1530/JME-16-0257 -
NLM
Nardelli TR, Vanzela EC, Benedicto KC, Brozzi F, Fujita A, Cardozo AK, Eizirik DL, Boschero AC, Ortis F. Prolactin protects against cytokine-induced beta-cell death by NFκB and JNK inhibition [Internet]. Journal of Molecular Endocrinology. 2018 ; 61( 1): 25-36.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1530/JME-16-0257 -
Vancouver
Nardelli TR, Vanzela EC, Benedicto KC, Brozzi F, Fujita A, Cardozo AK, Eizirik DL, Boschero AC, Ortis F. Prolactin protects against cytokine-induced beta-cell death by NFκB and JNK inhibition [Internet]. Journal of Molecular Endocrinology. 2018 ; 61( 1): 25-36.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1530/JME-16-0257 - Augmented β-Cell function and mass in glucocorticoid-treated rodents are associated with increased islet Ir-β/AKT/mTOR and decreased AMPK/ACC and AS160 signaling
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- Retículo endoplasmático e Complexo de Golgi
- Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation
- The non-canonical NF-κB pathway and its contribution to β-cell failure in diabetes
- Membranas celulares
- ARHGAP21 acts as an inhibitor of the glucose-stimulated insulin secretion process
Informações sobre o DOI: 10.1530/JME-16-0257 (Fonte: oaDOI API)
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