The relevance of kalikrein-kinin system via activation of B2 receptor in LPS-induced fever in rats (2017)
- Authors:
- USP affiliated authors: MELO, MIRIAM CRISTINA CONTIN DE - FCFRP ; SOUZA, GLORIA EMILIA PETTO DE - FCFRP
- Unidade: FCFRP
- DOI: 10.1016/j.neuropharm.2017.08.019
- Subjects: ANIMAIS; PROSTAGLANDINAS; NEURÔNIOS; FEBRE
- Keywords: Hypothalamic pre-optic area; LPS; Central mediation of fever; PGE2; B1 and B2 kinin receptors; Rats
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Neuropharmacology
- ISSN: 0028-3908
- Volume/Número/Paginação/Ano: v. 126, p. 84-96, 2017
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
SOARES, Denis de Melo et al. The relevance of kalikrein-kinin system via activation of B2 receptor in LPS-induced fever in rats. Neuropharmacology, v. 126, p. 84-96, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2017.08.019. Acesso em: 29 mar. 2024. -
APA
Soares, D. de M., Santos, D. R., Rummel, C., Ott, D., Melo, M. C. C. de, Roth, J., et al. (2017). The relevance of kalikrein-kinin system via activation of B2 receptor in LPS-induced fever in rats. Neuropharmacology, 126, 84-96. doi:10.1016/j.neuropharm.2017.08.019 -
NLM
Soares D de M, Santos DR, Rummel C, Ott D, Melo MCC de, Roth J, Calixto JB, Souza GEP de. The relevance of kalikrein-kinin system via activation of B2 receptor in LPS-induced fever in rats [Internet]. Neuropharmacology. 2017 ; 126 84-96.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1016/j.neuropharm.2017.08.019 -
Vancouver
Soares D de M, Santos DR, Rummel C, Ott D, Melo MCC de, Roth J, Calixto JB, Souza GEP de. The relevance of kalikrein-kinin system via activation of B2 receptor in LPS-induced fever in rats [Internet]. Neuropharmacology. 2017 ; 126 84-96.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1016/j.neuropharm.2017.08.019 - Antipyretic effect of ibuprofen on LPS-induced fever is not related to cytokines synthesis inhibition
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Informações sobre o DOI: 10.1016/j.neuropharm.2017.08.019 (Fonte: oaDOI API)
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