Reduced caveolae density in arteries of SHR contributes to endothelial dysfunction and ROS production (2019)
- Authors:
- USP affiliated authors: GRANDO, MARCELLA DARUGE - FCFRP ; BENDHACK, LUSIANE MARIA - FCFRP ; POTJE, SIMONE REGINA - FCFRP
- Unidade: FCFRP
- DOI: 10.1038/s41598-019-43193-8
- Subjects: ARTÉRIAS; MEMBRANA PLASMÁTICA; COLESTEROL; HIPERTENSÃO; ÓXIDO NÍTRICO
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Scientific Reports
- ISSN: 2045-2322
- Volume/Número/Paginação/Ano: v. 9, art. 6696, [16] p., 2019
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
POTJE, Simone Regina et al. Reduced caveolae density in arteries of SHR contributes to endothelial dysfunction and ROS production. Scientific Reports, v. 9, p. [16] , 2019Tradução . . Disponível em: https://doi.org/10.1038/s41598-019-43193-8. Acesso em: 16 abr. 2024. -
APA
Potje, S. R., Grando, M. D., Chignalia, A. Z., Antoniali, C., & Bendhack, L. M. (2019). Reduced caveolae density in arteries of SHR contributes to endothelial dysfunction and ROS production. Scientific Reports, 9, [16] . doi:10.1038/s41598-019-43193-8 -
NLM
Potje SR, Grando MD, Chignalia AZ, Antoniali C, Bendhack LM. Reduced caveolae density in arteries of SHR contributes to endothelial dysfunction and ROS production [Internet]. Scientific Reports. 2019 ; 9 [16] .[citado 2024 abr. 16 ] Available from: https://doi.org/10.1038/s41598-019-43193-8 -
Vancouver
Potje SR, Grando MD, Chignalia AZ, Antoniali C, Bendhack LM. Reduced caveolae density in arteries of SHR contributes to endothelial dysfunction and ROS production [Internet]. Scientific Reports. 2019 ; 9 [16] .[citado 2024 abr. 16 ] Available from: https://doi.org/10.1038/s41598-019-43193-8 - Endothelial cyclooxygenase differently modulates the contractile response to phenylephrine but not to thromboxane analog in aorta from normotensive and hypertensive rats
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Informações sobre o DOI: 10.1038/s41598-019-43193-8 (Fonte: oaDOI API)
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