Rho pathway: a fragile point in the GBM resistance to radiotherapy (2022)
- Authors:
- USP affiliated authors: FORTI, FÁBIO LUÍS - IQ ; MAGALHÃES, YULI THAMIRES - IQ
- Unidade: IQ
- Subjects: RADIOTERAPIA; NEOPLASIAS CEREBRAIS
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Bioquímica e Biologia Molecular
- Publisher place: São Paulo
- Date published: 2022
- Source:
- Título do periódico: Abstract Book
- Conference titles: Annual Meeting of the Brazilian Society for Biochemistry and Molecular Biology/SBBq
-
ABNT
MAGALHÃES, Yuli Thamires e FORTI, Fabio Luis. Rho pathway: a fragile point in the GBM resistance to radiotherapy. 2022, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular, 2022. Disponível em: https://www2.sbbq.org.br/reuniao/2022/images/livro_completo.pdf. Acesso em: 11 jun. 2024. -
APA
Magalhães, Y. T., & Forti, F. L. (2022). Rho pathway: a fragile point in the GBM resistance to radiotherapy. In Abstract Book. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular. Recuperado de https://www2.sbbq.org.br/reuniao/2022/images/livro_completo.pdf -
NLM
Magalhães YT, Forti FL. Rho pathway: a fragile point in the GBM resistance to radiotherapy [Internet]. Abstract Book. 2022 ;[citado 2024 jun. 11 ] Available from: https://www2.sbbq.org.br/reuniao/2022/images/livro_completo.pdf -
Vancouver
Magalhães YT, Forti FL. Rho pathway: a fragile point in the GBM resistance to radiotherapy [Internet]. Abstract Book. 2022 ;[citado 2024 jun. 11 ] Available from: https://www2.sbbq.org.br/reuniao/2022/images/livro_completo.pdf - ROCK inhibition reduces the sensitivity of mutant p53 glioblastoma to genotoxic stress through a Rac1-driven ROS production
- The Rho GTPase pathway as a potential chemosensitizer of TMZ and cisplatin-resistant GBM through a p53-dependent mechanism
- Glioblastoma: descoberto mecanismo capaz de reverter resistência do tumor cerebral à radioterapia
- Investigação do fenótipo de resistência múltipla a quimioterápicos em linhagens de glioblastoma humano
- Downregulation of the Rho GTPase pathway abrogates resistance to ionizing radiation in wild-type p53 glioblastoma by suppressing DNA repair mechanisms
- Exoenzyme C3 transferase lowers actin cytoskeleton dynamics, genomic stability and survival of malignant melanoma cells under UV-light stress
- GTPases, genome, actin: a hidden story in DNA damage response and repair mechanisms
- Effects DUSP3 silencing on cell proliferation and cell cycle regulators of xeroderma pigmentosum deficient cell lineages
- The RhoA GTPase mediate the repair pathways triggered by UV-promoted DNA lesions in mewo cells
- The RhoA and RhoB small GTPases mediate the repair signaling pathways triggered by UV-promoted DNA lesions
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