Assunto: FARMACOLOGIA
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CLAUDINO, Mário A. et al. Upregulation of gp91phox subunit of NAD(P)H oxidase contributes to erectile dysfunction caused by long-term nitric oxide inhibition in rats: reversion by regular physical training. Urology, v. 75, n. 4, p. 961-7, 2010Tradução . . Acesso em: 03 jun. 2024.APA
Claudino, M. A., Franco-Penteado, C. A., Priviero, F. B. M., Camargo, E. A., Teixeira, S. A., Muscará, M. N., et al. (2010). Upregulation of gp91phox subunit of NAD(P)H oxidase contributes to erectile dysfunction caused by long-term nitric oxide inhibition in rats: reversion by regular physical training. Urology, 75( 4), 961-7.NLM
Claudino MA, Franco-Penteado CA, Priviero FBM, Camargo EA, Teixeira SA, Muscará MN, De Nucci G, Zanesco A, Antunes E. Upregulation of gp91phox subunit of NAD(P)H oxidase contributes to erectile dysfunction caused by long-term nitric oxide inhibition in rats: reversion by regular physical training. Urology. 2010 ; 75( 4): 961-7.[citado 2024 jun. 03 ]Vancouver
Claudino MA, Franco-Penteado CA, Priviero FBM, Camargo EA, Teixeira SA, Muscará MN, De Nucci G, Zanesco A, Antunes E. Upregulation of gp91phox subunit of NAD(P)H oxidase contributes to erectile dysfunction caused by long-term nitric oxide inhibition in rats: reversion by regular physical training. Urology. 2010 ; 75( 4): 961-7.[citado 2024 jun. 03 ]